scispace - formally typeset
Search or ask a question

Showing papers on "Intracranial Lipoma published in 2000"


Journal Article
TL;DR: An unusually located intracranial lipoma was located deep in the left sylvian fissure, in the inferior parietal lobule, associated with cortical dysplasia of the surrounding supramarginal gyrus.
Abstract: We present the case of an unusually located intracranial lipoma in a 17-year-old patient with partial epilepsy who was being controlled with medication. The lipoma was located deep in the left sylvian fissure, in the inferior parietal lobule, associated with cortical dysplasia of the surrounding supramarginal gyrus. Abnormal vasculature was detected adjacent to and within the adipose mass. The findings of the imaging studies that included CT, MR imaging, and MR angiography, are described along with a brief review of the literature.

34 citations


Journal ArticleDOI
TL;DR: This work describes three patients with ENS, all of whom had in addition to the characteristic features of ENS intracranial and/or intraspinal lipomas, a typical finding of CNS lipoma, which caused a significant spastic movement disorder.
Abstract: Epidermal nevus syndrome (ENS) is a congenital neurocutaneous disorder characterized by linear nevus with a significant involvement of the nervous, ophthalmological and skeletal systems. Clinical manifestations of ENS include neurological features such as mental retardation, seizures, and movement disorders which are caused by a wide range of neuropathological lesions. We describe three patients with ENS, all of whom had in addition to the characteristic features of ENS intracranial and/or intraspinal lipomas. In one patient the lipoma extended from the thoracal vertebra 8 to the 4th ventricle; in the second patient it was localized on T9, and in the third patient an intracranial lipoma was located at the right cerebellopontine angle. The intraspinal lipomas caused a significant spastic movement disorder. So far, CNS lipomas have not been described as typical neuropathological findings in ENS. The differential diagnosis to encephalocraniocutaneous lipomatosis with the typical finding of CNS lipoma is discussed.

24 citations


Journal ArticleDOI
TL;DR: Two cases of a very rare tumor, intracranial lipoma, diagnosed by computed tomography (CT) and magnetic resonance imaging (MRI) are presented, one in the superior cerebellar cistern, the other in the periphery of the corpus callosum.
Abstract: We present two cases of a very rare tumor intracranial lipoma diagnosed by computed to‑ mography(CT)and magnetic resonance imaging(MRI). In one case the lipoma was in the su‑ perior cerebellar cistern the other was in the periphery of the corpus callosum. In the case in which MRI was used identification of the lipoma using a routine MRI examination was difficult. These cases are reported now because the incidental diagnosis of intracranial lipoma is likely to increase due to advanced neuroradiological techniques such as CT and MRI.(J Nippon Med Sch 2000; 67: 388―391)

17 citations


Journal ArticleDOI
TL;DR: It is concluded that surgery for CPA lipomas should be reserved for tumors that are truly symptomatic or if biopsy sampling is required for differential diagnosis, as was seen in this case.
Abstract: ed trigeminal neuralgia (TN). As seen in Fig. 1, magnetic resonance (MR) imaging of the cranium revealed a hyperintense mass lesion on both T1and T2-weighted images in the left cerebellopontine angle (CPA). A left suboccipital retrosigmoid approach disclosed a yellowish mass that encased the seventh and the eighth cranial nerves. The fifth cranial nerve was found to be displaced ventrally by the tumor. There were no vascular elements causing compression along the course of the trigeminal nerve. Approximately 40% of the tumor was removed but the seventh and the eighth cranial nerves were obviously traumatized. Postoperatively, the patient reported complete relief of TN but noted left-sided facial paralysis and deafness. Intracranial lipomas are uncommon; only 0.1% of brain lesions are fatty tumors.2 Of the 46 CPA lipomas reported, only seven cases have presented with TN.3 Lipomas usually occur at junctions between segments of the central nervous system, and these junctions represent sites of neural tube flexion as well as of redundant meninx primitiva.1 This finding supports the concept of lipoma formation as a result of abnormal persistence and maldifferentiation of the meninx. Surgery for CPA lipomas is quite controversial because of the difficulty of total removal of such a slow-growing lesion. Adherence to the adjacent cranial nerves is the major reason for increased postoperative morbidity, as was seen in our case.1,3 Malignant differentiation or rapid growth have never been reported.4 We conclude that surgery should be reserved for tumors that are truly symptomatic or if biopsy sampling is required for differential diagnosis.

15 citations