Showing papers on "Optic Disk Drusen published in 2007"

Hemorrhagic complications of drusen of the optic disk

Abstract: Introduction The hemorrhagic complications of the optic disk drusen are rare and interesting because of their functional implications, the difficulty of the differential diagnosis and the uncertain pathogeny. Material and methods: Retrospective study of 6 cases with a complete general and ophthalmological examination, followed between 2 and 9 years. Results The average age of the cases was 38.5 years (9-62 years). The visual acuity was slightly affected and reversible in the papillary hemorrhages (1 case), low and partial reversible in the extended hemorrhages (4 cases), severe affected and irreversible in the associations with a choroidal neovascular membrane with macular implications (1 case). We found three types of hemorrhages: papillary (1 case), peripapillary associated with choroidal neovascular membrane in acute (2 cases) and cicatricial stage (1 case), extended subretinal (2 cases). The papillary drusen were profound (4 cases) and superficial (2 cases). Ultrasonography showed the presence of the optic disk drusen in both eyes in all cases. The evolution followed on a period from 2 to 9 years, revealed the resorbtion of the hemorrhages between 3 and 6 months, the reversible visual acuity in the cases without macular implication, and the absence of recidiva or bilateralisation. Discussions and conclusions The authors are presenting a number of clinical cases of papillary drusen complicated with hemorrhages, some of the cases being associated with choroidial neovascular membrane. Because of the clinical appearance, aspects of differential diagnosis with papillary edema of other etiologies are discussed. A role in the pathogeny is attributed to the direct or progressive compression of the retinal vascularisation which can provoke the erosion of the vessels and in some cases the appearance of the choroidal neovascular membrane. The suggested therapy is medical, surgical, or by photocoagulation, but the results are contradictory.

Transient monocular visual loss.

Abstract: Transient monocular visual loss may be caused by a variety of ophthalmic and systemic conditions. Management depends on identifying the cause. Embolic occlusions of arteries supplying the eye are a most important cause. Such emboli often arise from the heart, aorta, and internal carotid arteries. The most common embolic substances are white platelet-fibrin and red erythrocyte-fibrin thrombi, cholesterol crystals, and calcific particles. Retinal vasoconstriction is another important cause of transient monocular visual loss. The conditions that cause anterior ischemic optic neuropathy may occasionally cause transient visual loss. Transient monocular visual loss may also be related to conditions that cause papilledema and some conditions intrinsic to the eye, such as optic disk drusen and colobomas.

Acute anterior ischemic optic neuropathy in the course of optic nerve drusen

Abstract: Authors present the case of 48 years old male with acute anterior ischemic optic neuropathy (AION) in presence of optic nerve drusen. AION occurred in both eyes, but not at the same time. Diagnosis was made on the basis of laboratory tests, ultrasonographic examination and fluorescein angiography. In the course of treatment improvement of visual acuity was achieved , but concentric constriction of visual field remained. As no effective treatment of non vascular AION was proposed so far, major stress should be put on elimination of its vascular risk factors.