A
A.A. van Kraats
Researcher at University of Amsterdam
Publications - 4
Citations - 83
A.A. van Kraats is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Renal blood flow & Renal function. The author has an hindex of 4, co-authored 4 publications receiving 83 citations.
Papers
More filters
Journal ArticleDOI
High-energy phosphates in heart, liver, kidney, and skeletal muscle of endotoxemic rats
TL;DR: In anesthetized endotoxemic rats, organ perfusion and the storage of high-energy phosphates of heart, liver, kidney, and skeletal muscle are studied and the effects of improving cardiac output (CO) and organ blood flow with cardiostimulatory drugs [dopexamine (DX) and dobutamine (DB)].
Journal Article
Renal function and metabolism during endotoxemia in rats: role of hypoperfusion.
A.A. van Lambalgen,A.A. van Kraats,G. C. Van Den Bos,H.V. Stel,J.P. Straub,A.J.M. Donker,Lambertus G. Thijs +6 more
TL;DR: Renal function and metabolism was more affected in this hypodynamic form of endotoxin shock than in "balloon shock," which may be caused by the effects of endotoxemia on sticking of leukocytes and renal metabolism.
Journal ArticleDOI
Development of renal failure in endotoxemic rats : can it be explained by early changes in renal energy metabolism ?
A.A. van Lambalgen,A.A. van Kraats,G. C. Van Den Bos,T. Teerlink,H.V. Stel,A.J.M. Donker,Lambertus G. Thijs +6 more
TL;DR: Testing the hypothesis that early changes in renal metabolism contribute to the development of acute renal failure during endotoxin shock found no systemic hemodynamic or biochemical changes.
Journal ArticleDOI
Systemic and renal actions of dopexamine and dobutamine during endotoxin shock in the rat
A.A. van Lambalgen,A.A. van Kraats,M.F. van de Vaart-Mulder,G. C. Van Den Bos,H.V. Stel,J.P. Straub,A.J.M. Donker,Lambertus G. Thijs +7 more
TL;DR: The major action of dopamine and β2 receptor agonist dopexamine seems to be an increase in RPF that cannot be attributed to restored CO alone, but also to decreased renal vascular resistance.