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Agnes Michalczyk

Researcher at Centre for Cellular and Molecular Biology

Publications -  44
Citations -  1288

Agnes Michalczyk is an academic researcher from Centre for Cellular and Molecular Biology. The author has contributed to research in topics: Zinc & Nostoc punctiforme. The author has an hindex of 21, co-authored 41 publications receiving 1166 citations. Previous affiliations of Agnes Michalczyk include Deakin University.

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Zinc deficiency and its inherited disorders -a review.

TL;DR: Taking into account the fact that there are no definitive tests for zinc deficiency and that this disorder can go undiagnosed, plus the recent identification of multiple members of the SCL30 and SLC39, it is likely that mutations in other genes may underlie additional inherited disorders of zinc deficiency.
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Copper is taken up efficiently from albumin and α2-macroglobulin by cultured human cells by more than one mechanism

TL;DR: The proteins mainly responsible for the plasma-exchangeable copper pool deliver the metal to mammalian cells efficiently and by several different mechanisms are concluded.
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Zinc and infant nutrition.

TL;DR: The significance of adequate zinc nutrition in infants, factors that influence zinc nutrition, the consequences of zinc deficiency, including its contribution to the global burden of disease, and addresses some of the knowledge gaps in zinc biology are discussed.
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Lubricin: a versatile, biological anti-adhesive with properties comparable to polyethylene glycol

TL;DR: It is shown that coatings of lubricin protein are as effective as, or better than, self-assembled monolayers of polyethylene glycol over a wide range of pH and that this provides a simple, versatile, highly stable, and highly effective method of controlling unwanted adhesion to surfaces.
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Defective localization of the Wilson disease protein (ATP7B) in the mammary gland of the toxic milk mouse and the effects of copper supplementation.

TL;DR: The impaired copper transport from the mammary gland into milk in lactating tx mice is related to the mislocalization of ATP7B, which would explain the inability of the tx mouse to secrete normal amounts of copper in milk.