A
Akira Fujiki
Researcher at University of Toyama
Publications - 119
Citations - 2628
Akira Fujiki is an academic researcher from University of Toyama. The author has contributed to research in topics: Atrial fibrillation & Fibrillation. The author has an hindex of 28, co-authored 116 publications receiving 2485 citations.
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Journal ArticleDOI
Prevalence of atrial fibrillation in the general population of Japan: an analysis based on periodic health examination.
Hiroshi Inoue,Akira Fujiki,Hideki Origasa,Satoshi Ogawa,Ken Okumura,Isao Kubota,Yoshifusa Aizawa,Takeshi Yamashita,Hirotsugu Atarashi,Minoru Horie,Tohru Ohe,Yoshinori Doi,Akihiko Shimizu,Akiko Chishaki,Tetsunori Saikawa,Katsusuke Yano,Akira Kitabatake,Hideo Mitamura,Itsuo Kodama,Shiro Kamakura +19 more
TL;DR: The prevalence of AF increased in Japan as the population aged, as in Western countries, and the overall prevalence in Japan is approximately two-thirds of that in the USA.
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Vagal Activity Modulates Spontaneous Augmentation of ST Elevation in the Daily Life of Patients with Brugada Syndrome
Koichi Mizumaki,Akira Fujiki,Takayuki Tsuneda,Masao Sakabe,Kunihiro Nishida,Masataka Sugao,Hiroshi Inoue +6 more
TL;DR: The aim of the present study was to elucidate the relation between spontaneous augmentation of ST elevation and changes in autonomic nervous activities in the daily life of patients with Brugada syndrome.
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ST segment elevation in the right precordial leads induced with class IC antiarrhythmic drugs: insight into the mechanism of Brugada syndrome.
TL;DR: Two patients without previous episodes of syncope who showed characteristic ECG changes similar to Brugada syndrome following administration of Class IC drugs, flecainide and pilsicainide were evaluated.
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Usefulness and safety of bepridil in converting persistent atrial fibrillation to sinus rhythm
TL;DR: Bepridil is effective and safe for terminating long-lasting persistent AF and restored sinus rhythm alone or in combination with aprindine in 69% of 32 patients with persistent AF lasting > or = 3 months.
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Enalapril Prevents Perpetuation of Atrial Fibrillation by Suppressing Atrial Fibrosis and Over-Expression of Connexin43 in a Canine Model of Atrial Pacing-Induced Left Ventricular Dysfunction
Masao Sakabe,Akira Fujiki,Kunihiro Nishida,Masataka Sugao,Hidehiko Nagasawa,Takayuki Tsuneda,Koichi Mizumaki,Hiroshi Inoue +7 more
TL;DR: Enalapril suppressed atrial pacing-induced AF with tachycardia-mediated cardiomyopathy by suppressing interstitial fibrosis, connexin43 over-expression and conduction delay.