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Alicia Beatriz Motta
Researcher at University of Buenos Aires
Publications - 83
Citations - 1644
Alicia Beatriz Motta is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Polycystic ovary & Nitric oxide. The author has an hindex of 23, co-authored 80 publications receiving 1444 citations. Previous affiliations of Alicia Beatriz Motta include National Scientific and Technical Research Council.
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The mechanisms involved in the action of metformin in regulating ovarian function in hyperandrogenized mice.
Evelin Mariel Elia,Valeria A. Sander,Carolina Griselda Luchetti,Maria Emilia Solano,G. Di Girolamo,Claudio Gonzalez,Alicia Beatriz Motta +6 more
TL;DR: Investigation of the mechanisms by which N,N'-dimethylbiguanide metformin prevents the ovarian disorders provoked by the hyperandrogenization with dehydroepiandrosterone (DHEA) in prepuberal BALB/c mice found it to prevent the increase of ovarian iNOS and COX2 expressions and to enhance the activation of phosphorylated AMPK-alpha expression.
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Effects of dehydroepiandrosterone on ovarian cystogenesis and immune function
Carolina Griselda Luchetti,Maria Emilia Solano,Valeria A. Sander,María Laura Barreiro Arcos,Claudio Gonzalez,Guillermo Di Girolamo,Sara Chiocchio,Graciela Cremaschi,Alicia Beatriz Motta +8 more
TL;DR: Results show that the development of cysts involves changes in ovarian function and an imbalance in the oxidant-antioxidant equilibrium.
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The role of obesity in the development of polycystic ovary syndrome
TL;DR: It has been reported that, after losing as little as 5 % of initial body weight obese women with PCOS improved spontaneous ovulation rates and spontaneous pregnancy, therefore, the weight loss prior to conception improves live birth rate in obesity women with or without PCOS.
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Role of the N, N'-dimethylbiguanide metformin in the treatment of female prepuberal BALB/c mice hyperandrogenized with dehydroepiandrosterone.
Valeria A. Sander,Carolina Griselda Luchetti,Maria Emilia Solano,Evelin Mariel Elia,Guillermo Di Girolamo,Claudio Gonzalez,Alicia Beatriz Motta +6 more
TL;DR: Results indicate that metformin is able, directly or indirectly, to avoid the endocrine and immune alterations produced when mice are hyperandrogenized with DHEA.
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Impaired immune responses in streptozotocin-induced type I diabetes in mice. Involvement of high glucose.
TL;DR: Results indicate that HG concentrations can directly affect lymphoid cell growth and suggest that prolonged exposure to pathologically HG concentrations would result in the immunosuppressive state observed in diabetes.