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Andreas Bonz
Researcher at University of Würzburg
Publications - 26
Citations - 1100
Andreas Bonz is an academic researcher from University of Würzburg. The author has contributed to research in topics: Clopidogrel & Tirofiban. The author has an hindex of 14, co-authored 26 publications receiving 1045 citations.
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Journal ArticleDOI
Cannabinoids acting on CB1 receptors decrease contractile performance in human atrial muscle.
Andreas Bonz,Martin Laser,Sina Küllmer,Silke Kniesch,Jörg Babin-Ebell,Verena Popp,Georg Ertl,Jens A. Wagner +7 more
TL;DR: The conclusion is that anandamide, R-methanandamide and HU-210 decrease contractile performance in human atrial muscle via CB1 receptors.
Journal ArticleDOI
effect of additional temporary glycoprotein IIb/IIIa receptor inhibition on troponin release in elective percutaneous coronary interventions after pretreatment with aspirin and clopidogrel (TOPSTAR trial)
Andreas Bonz,Björn Lengenfelder,Jörg Strotmann,Stefanie Held,Oliver Turschner,K Harre,Christian M. Wacker,Christiane Waller,Nikolaus Kochsiek,Malte Meesmann,Ludwig Neyses,P. Schanzenbächer,Georg Ertl,Wolfram Voelker +13 more
TL;DR: The GP IIb/IIIa receptor antagonist tirofiban is able to decrease the incidence of troponin release significantly in this patient population and after nine months a reduction of combined death/myocardial infarction/target vessel revascularization could be observed in the tiroFiban group.
Journal ArticleDOI
Conditional Neuronal Nitric Oxide Synthase Overexpression Impairs Myocardial Contractility
Natalie Burkard,Adam G. Rokita,Susann G. Kaufmann,Matthias Hallhuber,Rongxue Wu,Kai Hu,Ulrich Hofmann,Andreas Bonz,Stefan Frantz,Elizabeth J. Cartwright,Ludwig Neyses,Lars S. Maier,Sebastian Maier,Thomas Renné,Kai Schuh,Oliver Ritter +15 more
TL;DR: It is suggested that nNOS might suppress the function of L-type Ca2+-channels and in turn reduces Ca2-transients which accounts for the negative inotropic effect.
Journal ArticleDOI
Myosin Light Chain–Actin Interaction Regulates Cardiac Contractility
TL;DR: In this article, the amino-terminal domain of the essential myosin light chain (MLC-1) binds to the carboxy terminus of the actin molecule.
Journal ArticleDOI
The estrogen receptor-alpha agonist 16alpha-LE2 inhibits cardiac hypertrophy and improves hemodynamic function in estrogen-deficient spontaneously hypertensive rats
Theo Pelzer,Virginija Jazbutyte,Kai Hu,Stephan Segerer,Matthias Nahrendorf,Peter Nordbeck,Andreas Bonz,Jenny Muck,K. H. Fritzemeier,Christa Hegele-Hartung,Georg Ertl,Ludwig Neyses +11 more
TL;DR: The hypothesis that activation of ERalpha favorably affects cardiac hypertrophy, myocardial contractility, and gene expression in ovariectomized SHR is supported, and further studies are required to determine whether activation ERbeta mediates redundant or divergent effects.