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Angelina Rakovska

Researcher at Bulgarian Academy of Sciences

Publications -  17
Citations -  513

Angelina Rakovska is an academic researcher from Bulgarian Academy of Sciences. The author has contributed to research in topics: Cholinergic & Acetylcholine. The author has an hindex of 11, co-authored 17 publications receiving 485 citations.

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Effects of novelty and habituation on acetylcholine, GABA, and glutamate release from the frontal cortex and hippocampus of freely moving rats.

TL;DR: ACh released by the animal placed in a novel environment seems to have two components, one related to motor activity and onerelated to attention, anxiety and fear, which disappears in the familiar environment, where ACh release is directly related toMotor activity.
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Neurotensin modulation of acetylcholine and gaba release from the rat hippocampus: an in vivo microdialysis study

TL;DR: These findings indicate for the first time that neurotensin plays a neuromodulatory role in the regulation of GABAergic and cholinergic neuronal activity in the hippocampus of awake and freely moving rats.
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Neurotensin modulation of acetylcholine, GABA, and aspartate release from rat prefrontal cortex studied in vivo with microdialysis

TL;DR: In vivo NT plays a modulatory role in the PFC by interacting with cortical neurons releasing GABA and Asp and with ACh-containing neurons projecting to the P FC, and mediated by the NTR1 receptor.
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Activation of non-NMDA receptors stimulates acetylcholine and GABA release from dorsal hippocampus: a microdialysis study in the rat.

TL;DR: The results demonstrate that non-NMDA glutamatergic receptors in the hippocampus regulate hippocampal release of GABA and ACh.
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Physiological release of striatal acetylcholine (in vivo): effect of somatostatin on dopaminergic-cholinergic interaction

TL;DR: The findings indicate that SOM is capable of releasing both ACh and DA in the striatum, however, its effect on ACh release is partially masked unless the D(2) receptor-mediated tonic inhibitory effect of released DA from the nigro-striatal pathway is attenuated.