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Anjali Rani

Researcher at Institute of Medical Sciences, Banaras Hindu University

Publications -  39
Citations -  198

Anjali Rani is an academic researcher from Institute of Medical Sciences, Banaras Hindu University. The author has contributed to research in topics: Pregnancy & Medicine. The author has an hindex of 6, co-authored 34 publications receiving 107 citations. Previous affiliations of Anjali Rani include Banaras Hindu University.

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Unsafe abortion and abortion-related death among 1.8 million women in India.

TL;DR: Despite abortion being legal, the high estimated prevalence of unsafe abortion demonstrates a major public health problem in India as Socioeconomic vulnerability and inadequate access to healthcare services combine to leave large numbers of women at risk of unsafeabortion and abortion-related death.
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Stillbirth among women in nine states in India: rate and risk factors in study of 886,505 women from the annual health survey

TL;DR: India is an emerging market economy experiencing a rapid health transition, yet these findings demonstrate the marked disparity in risk of stillbirth by women’s socioeconomic status.
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Breastfeeding Knowledge and Behavior Among Women Visiting a Tertiary Care Center in India: A Cross-Sectional Survey.

TL;DR: Although breastfeeding is practiced by a majority of mothers in a developing country like India, there is a significant gap in knowledge and optimal breastfeeding behaviors, which can be encouraged by proper counseling by health care workers and organizing educational programs focusing women especially with low education and limited resources.
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Risk factors for maternal mortality among 1.9 million women in nine empowered action group states in India: secondary analysis of Annual Health Survey data.

TL;DR: Maternal mortality in India’s EAG states greatly exceeds the national average and the study showed that the risk conferred by poor socioeconomic status could be mitigated by universal access to healthcare during pregnancy and childbirth.
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Deoxyelephantopin, a novel naturally occurring phytochemical impairs growth, induces G2/M arrest, ROS-mediated apoptosis and modulates lncRNA expression against uterine leiomyoma.

TL;DR: DOE inhibits growth of UL cells via cell cycle arrest at G2/M phase, induces ROS-dependent caspase-3-mediated mitochondrial intrinsic apoptotic pathway and down-regulation of oncogenic lncRNA in UL cells.