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Ashok Kumar Tripathi

Researcher at University College of Medical Sciences

Publications -  48
Citations -  1225

Ashok Kumar Tripathi is an academic researcher from University College of Medical Sciences. The author has contributed to research in topics: Oxidative stress & Kidney disease. The author has an hindex of 22, co-authored 47 publications receiving 1057 citations. Previous affiliations of Ashok Kumar Tripathi include Indian Institutes of Technology.

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Protective effects of dietary ginger (Zingiber officinales Rosc.) on lindane-induced oxidative stress in rats.

TL;DR: It is suggested that a diet containing naturally occurring compounds is effective in exerting protective effects by modulating oxidative stress in rats fed a normal diet.
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Low intensity microwave radiation induced oxidative stress, inflammatory response and DNA damage in rat brain

TL;DR: It is suggested thatLow intensity microwave radiation induces oxidative stress, inflammatory response and DNA damage in brain by exerting a frequency dependent effect and increased oxidative stress and inflammatory response might be the factors involved in DNA damage following low intensity microwave exposure.
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Advanced glycation end products enhance reactive oxygen and nitrogen species generation in neutrophils in vitro

TL;DR: Evidence is provided that AGEs may play a key role in the induction of oxidative stress through the augmentation of PMN-mediated ROS and RNI generation and this may be in part responsible for development of AGE-induced diabetic pathology.
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Role of advanced glycation end product (AGE)-induced receptor (RAGE) expression in diabetic vascular complications.

TL;DR: Serum AGE level was significantly higher in diabetic patients having vascular complications as compared to T2DM without complications and RAGE m-RNA expression level in PBMCs assayed by quantitative real time PCR was four timesHigher in diabetic subjects without vascular complications.
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A study on serum advanced glycation end products and its association with oxidative stress and paraoxonase activity in type 2 diabetic patients with vascular complications.

TL;DR: High serum AGE concentration and low PON1 activity may be considered as additional risk factor for development of vascular complications in type 2 DM (T2DM) in diabetic subjects.