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Atsuo Suzuki
Researcher at Nagoya University
Publications - 24
Citations - 161
Atsuo Suzuki is an academic researcher from Nagoya University. The author has contributed to research in topics: Mutant & Missense mutation. The author has an hindex of 8, co-authored 24 publications receiving 122 citations.
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Journal ArticleDOI
Impaired secretion of carboxyl-terminal truncated factor VII due to an F7 nonsense mutation associated with FVII deficiency
Ryoko Tanaka,Daisuke Nakashima,Atsuo Suzuki,Yuhri Miyawaki,Yuta Fujimori,Takayuki Yamada,Akira Takagi,Akira Takagi,Takashi Murate,Takashi Murate,Koji Yamamoto,Akira Katsumi,Tadashi Matsushita,Tomoki Naoe,Tetsuhito Kojima +14 more
TL;DR: The carboxyl-terminal amino acids of FVII play an important role in its secretion, and the p.Arg462X mutation was likely to have caused the FVII deficiency in this patient.
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Clot waveform analysis in Clauss fibrinogen assay contributes to classification of fibrinogen disorders.
Atsuo Suzuki,Nobuaki Suzuki,Takeshi Kanematsu,Sho Shinohara,Nobuo Arai,Ryosuke Kikuchi,Tadashi Matsushita +6 more
TL;DR: The CWA of CFA could distinguish fibrinogen disorders using a combination of Ac/Min1 and Ac/eAg values, and allows the qualitative detection of fibr inogen disorder easily and represents a novel screening test for fibrInogen disorders.
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Association between the ratio of anti-angiogenic isoform of VEGF-A to total VEGF-A and adverse clinical outcomes in patients after acute myocardial infarction.
Kazuhiro Harada,Ryosuke Kikuchi,Hideki Ishii,Yohei Shibata,Susumu Suzuki,Akihito Tanaka,Atsuo Suzuki,Kenshi Hirayama,Toyoaki Murohara +8 more
TL;DR: The assessment of VEGf-A165b combined with VEGF-A may be a valuable screening tool for predicting MACCEs in clinical practice.
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Balance between angiogenic and anti-angiogenic isoforms of VEGF-A is associated with the complexity and severity of coronary artery disease.
Yohei Shibata,Ryosuke Kikuchi,Hideki Ishii,Susumu Suzuki,Kazuhiro Harada,Kenshi Hirayama,Atsuo Suzuki,Yosuke Tatami,Kazuhisa Kondo,Toyoaki Murohara +9 more
TL;DR: Evaluating the relationship between the complexities of CAD using the Syntax score (SS) and the levels of circulating total VEGF-A or VEGf-A165b in patients with complex CAD indicated that high SS was associated with circulating levels of total VEL-A and the ratio of VEGFs-A-165b to total VES-A in patientsWith complex CAD.
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A novel missense mutation causing abnormal LMAN1 in a Japanese patient with combined deficiency of factor V and factor VIII
Takayuki Yamada,Yuta Fujimori,Atsuo Suzuki,Yuhri Miyawaki,Akira Takagi,Akira Takagi,Takashi Murate,Takashi Murate,Masayuki Sano,Tadashi Matsushita,Hidehiko Saito,Tetsuhito Kojima,Tetsuhito Kojima +12 more
TL;DR: It was suggested that the p.Trp67Ser mutation might affect the molecular chaperone function of LMAN1, impairing affinity for D‐mannose as well as for MCFD2, which may be responsible for F5F8D in the patient.