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Axu Hu
Researcher at Minzu University of China
Publications - 7
Citations - 68
Axu Hu is an academic researcher from Minzu University of China. The author has contributed to research in topics: Mismatch negativity & Lateral inhibition. The author has an hindex of 2, co-authored 4 publications receiving 53 citations. Previous affiliations of Axu Hu include Northwest University for Nationalities.
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Left hemisphere lateralization for lexical and acoustic pitch processing in Cantonese speakers as revealed by mismatch negativity
TL;DR: The mismatch negativity elicited by lexical pitch contrast was lateralized to the left hemisphere, which is consistent with the pattern of function-dependent brain asymmetry (i.e., left hemisphere lateralization for speech processing) in nontonal language speakers.
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Lateral Inhibition is a Neural Mechanism Underlying Mismatch Negativity.
TL;DR: An oddball paradigm was developed in which tone-pairs composed of two sinusoidal tones were presented as standards and deviants, and results suggest that lateral inhibition is a neural mechanism that underlies the MMN response.
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Visual mismatch negativity elicited by semantic violations in visual words
TL;DR: The semantic radical, which has a high frequency of occurrence because it is carried by many words, may be critical for the elicitation of vMMN, the visual counterpart of auditory MMN.
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A lateral inhibition mechanism explains the dissociation between mismatch negativity and behavioral pitch discrimination.
TL;DR: The results support the notion that an enhanced lateral inhibition mechanism underlies superior pitch discrimination, a critical neural mechanism that sharpens the tuning curves of the auditory neurons in the tonotopy.
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Investigating app icon recognition with event-related potentials
TL;DR: In this article , the authors present familiar and unfamiliar app icons and ask participants to perform a repetition detection task while recording the event-related potentials elicited by these stimuli, and find that the recognition of familiar app icons results in the activation of the ventral occipitotemporal cortex approximately 220 ms after exposure.