B
B J Rathbone
Publications - 5
Citations - 1049
B J Rathbone is an academic researcher. The author has contributed to research in topics: Gastritis & Helicobacter pylori. The author has an hindex of 5, co-authored 5 publications receiving 1026 citations.
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Campylobacter pyloridis and acid induced gastric metaplasia in the pathogenesis of duodenitis.
TL;DR: The results suggest that acid induced gastric metaplasia in the duodenum and C pyloridis associated gastritis may be synergistic in the pathogenesis of duodenitis; the metaplastic gastric epithelium allows C plyoridis to colonise theDuodenal mucosa, where it produces an acute inflammatory response.
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Bacterial adhesion and disease activity in Helicobacter associated chronic gastritis.
S. J. Hessey,J. Spencer,Judith I. Wyatt,G. M. Sobala,B J Rathbone,A. T. R. Axon,Michael F. Dixon +6 more
TL;DR: A significant increase in the proportion of attached bacteria in cases showing histological epithelial degeneration, and a significant decrease in cases show intraepithelial polymorph infiltration are found.
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Local immune response to gastric Campylobacter in non-ulcer dyspepsia.
TL;DR: IgA coated bacteria were seen in all cases of active gastritis, and in 60% of biopsy specimens without intraepithelial neutrophils, while coating with IgG or IgM, or both, was correlated with activity of gastritis and was rarely seen in the absence of a neutrophil infiltrate.
Journal ArticleDOI
Gastric epithelium in the duodenum: its association with Helicobacter pylori and inflammation.
Judith I. Wyatt,B J Rathbone,G. M. Sobala,T M Shallcross,Richard V. Heatley,A. T. R. Axon,Michael F. Dixon +6 more
TL;DR: It is proposed that inflammatory injury to the duodenal mucosa by H pylori may stimulate the development of further gastric metaplasia, and that the area of duodenum susceptible to colonisation with H plyori may increase progressively until mucosal integrity is compromised and ulceration supervenes.
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Helicobacter pylori serology in patients with coeliac disease and dermatitis herpetiformis
Jean E. Crabtree,S O'Mahony,Judith I. Wyatt,R V Heatley,J P Vestey,P D Howdle,B J Rathbone,M S Losowsky +7 more
TL;DR: In this article, the authors investigate whether Helicobacter pylori infection or autoimmune gastritis is responsible for the reported increase in gastric pathology and abnormalities of gastric function in patients with coeliac disease and dermatitis herpetiformis (DH).