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Benedetto N
Researcher at Seconda Università degli Studi di Napoli
Publications - 5
Citations - 53
Benedetto N is an academic researcher from Seconda Università degli Studi di Napoli. The author has contributed to research in topics: Toxoplasma gondii & Endothelial stem cell. The author has an hindex of 4, co-authored 5 publications receiving 51 citations.
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Journal Article
Effect of prolactin, rIFN-gamma or rTNF-alpha in murine toxoplasmosis.
TL;DR: It is concluded that prolactin may play an important role in modulating the host's immune defence against T. gondii opportunistic infection and that PRL can regulate in vivo endogenous TNF-alpha production in the cytokine cascade.
Journal Article
Effects of alpha-adrenergic agonists on Toxoplasma gondii replication in human umbilical vein endothelial cells.
TL;DR: The results raise the interesting possibility that alpha 2 and beta-adrenergics agonists, which naturally occur in body fluids, may regulate the transplacental transmission of T. gondii from mother to foetus.
Journal Article
Susceptibility to toxoplasmosis: correlation between macrophage function, brain cyst formation and mortality in rats.
TL;DR: The results suggest that the susceptibility of drug-immunosuppressed rats is not due exclusively to a deficient macrophage function, but is probably also linked to immune mechanisms involved in the process of cystogenesis.
Journal Article
Interactions between cytokines and growth hormone for resistance to experimental.
TL;DR: The reduction of intracellular killing by monoclonal antibodies to ICAM-1 suggest that this adhesin plays a role in controlling T. gondii entry into cells, and data indicate that NO2- itself does not directly affect toxoplasmastatic activity.
Journal Article
Impairment of natural resistance to Toxoplasma gondii infection in rats treated with beta adrenergics, beta blockers, corticosteroids or total body irradiation.
Benedetto N,Folgore A,Galdiero M +2 more
TL;DR: It is hypothesised that in rats the natural resistance to T. gondii can be modulate beta adrenergics or corticosteroids and do not significantly differ from untreated rats in their time of survival.