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Bernhard Tillmann
Researcher at University of Kiel
Publications - 128
Citations - 5514
Bernhard Tillmann is an academic researcher from University of Kiel. The author has contributed to research in topics: Cartilage & Vascular endothelial growth factor. The author has an hindex of 43, co-authored 127 publications receiving 5144 citations.
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Journal ArticleDOI
Collagenous fibril texture of the human knee joint menisci.
Wolf Petersen,Bernhard Tillmann +1 more
TL;DR: The circular arrangement of collagen fibrils in the central portion of the meniscus provides a functional explanation for the longitudinal orientation of the majority of tears in the menisci tissue.
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The role of vasculature and angiogenesis for the pathogenesis of degenerative tendons disease.
TL;DR: Avascularity is considered to be a key factor for the etiology of degenerative tendon disease, and the repair capability after repetitive microtrauma is strongly compromised in avascular tissue of gliding tendons.
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Structure and vascularization of the cruciate ligaments of the human knee joint
Wolf Petersen,Bernhard Tillmann +1 more
TL;DR: The structure and vascularization of the human anterior and posterior cruciate ligament were investigated by light microscope, transmission electron microscopy, injection techniques and by immunohistochemistry to reveal typical features of chondrocytes.
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Curving and looping of the internal carotid artery in relation to the pharynx: frequency, embryology and clinical implications.
TL;DR: Variations of the internal carotid artery leading to direct contact with the pharyngeal wall are likely to be of great clinical relevance in view of the large number of routine procedures performed and may be of significance in relation to the occurrence of cerebrovascular symptoms.
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The splice variants VEGF121 and VEGF189 of the angiogenic peptide vascular endothelial growth factor are expressed in osteoarthritic cartilage.
TL;DR: Both proteins should contribute to the inflammatory process by autocrine/paracrine stimulation of chondrocytes, chemotaxis of macrophages, and promotion of angiogenesis.