Bianca M. Cattaneo

TL;DR: In this paper, the authors investigated whether these alterations involve sympathetic drive in 10 young obese sub-groups and found that the sympathetic drive was not involved in the majority of the cases.

TL;DR: In both essential and secondary hypertensives, baroreceptor-heart rate control was displaced toward elevated blood pressure values and markedly impaired compared with normotensive subjects, and sympathetic activation characterizes essential but not secondary hypertension.

TL;DR: Reduction in body weight induced by a hypocaloric diet with normal sodium content exerts a marked reduction in sympathetic activity owing to central sympathoinhibition, which can be due to the consequences of an increased insulin sensitivity but also to a restoration of the baroreflex control of the cardiovascular system with weight loss.

TL;DR: These results provide the first direct evidence that in congestive heart failure chronic ACE inhibitor treatment is accompanied by a marked reduction in central sympathetic outflow, and may depend on a persistent restoration of baroreflex restraint on the sympathetic neural drive.

TL;DR: Evidence is raised that in humans sodium restriction may impair the arterial baroreflex, which may be responsible for the sympathetic activation occurring in this condition and for the impairment of blood pressure homeostasis.