B
Börje Haraldsson
Researcher at University of Gothenburg
Publications - 129
Citations - 7103
Börje Haraldsson is an academic researcher from University of Gothenburg. The author has contributed to research in topics: Peritoneal dialysis & Renal function. The author has an hindex of 41, co-authored 128 publications receiving 6550 citations. Previous affiliations of Börje Haraldsson include Sahlgrenska University Hospital.
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Journal ArticleDOI
Properties of the Glomerular Barrier and Mechanisms of Proteinuria
TL;DR: The cellular components are the key players in restricting solute transport, while the GBM is responsible for most of the resistance to water flow across the glomerular barrier.
Journal ArticleDOI
Transport of macromolecules across microvascular walls: the two-pore theory
Bengt Rippe,Börje Haraldsson +1 more
TL;DR: This review summarized the evidence favoring the concept that the major plasma proteins are passively transported across vascular walls through water-filled pathways by means of convection and diffusion and discussed the general behavior of such a heteroporous system.
Journal ArticleDOI
Computer simulations of peritoneal fluid transport in CAPD.
TL;DR: It is suggested that computer modelling based on a three-pore model of membrane selectivity may be a useful tool for describing the IPV versus time relationships under various conditions in CAPD.
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Long-term clinical effects of a peritoneal dialysis fluid with less glucose degradation products
Bengt Rippe,Ole Simonsen,Olle Heimbürger,Anders Christensson,Börje Haraldsson,Gunnar Stelin,Lars Weiss,Finn.-David. Nielsen,Susanne Bro,Michael Friedberg,Anders Wieslander +10 more
TL;DR: The results indicate that the new solution causes less mesothelial and interstitial damage than conventional ones; that is, it may be considered more biocompatible than a number of conventional PD solutions currently in use.
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Endothelial mitochondrial oxidative stress determines podocyte depletion in segmental glomerulosclerosis
Ilse Daehn,Gabriella Casalena,Taoran Zhang,Shaolin Shi,Franz Fenninger,Nicholas Barasch,Liping Yu,Vivette D. D'Agati,Detlef Schlöndorff,Wilhelm Kriz,Börje Haraldsson,Erwin P. Bottinger +11 more
TL;DR: These studies indicate that segmental glomerulosclerosis develops as a result of podocyte-endothelial crosstalk mediated by EDN1/EDNRA-dependent mitochondrial dysfunction and suggest that targeting the reciprocal interaction between podocytes and endothelia may provide opportunities for therapeutic intervention in FSGS.