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Bradley J. Hindman

Researcher at University of Iowa

Publications -  132
Citations -  4642

Bradley J. Hindman is an academic researcher from University of Iowa. The author has contributed to research in topics: Cardiopulmonary bypass & Cerebral blood flow. The author has an hindex of 37, co-authored 125 publications receiving 4341 citations. Previous affiliations of Bradley J. Hindman include University of Iowa Hospitals and Clinics & Saint Barnabas Medical Center.

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Mild Intraoperative Hypothermia during Surgery for Intracranial Aneurysm

TL;DR: Intraoperative hypothermia did not improve the neurologic outcome after craniotomy among good-grade patients with aneurysmal subarachnoid hemorrhage.
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A prospective, comparative trial of three anesthetics for elective supratentorial craniotomy. Propofol/fentanyl, isoflurane/nitrous oxide, and fentanyl/nitrous oxide.

TL;DR: Compared three anesthetic techniques in 121 adults undergoing elective surgical removal of a supratentorial, intracranial mass lesion indicate that, despite their respective cerebrovascular effects, all of the anesthetic regimens used were acceptable in these patients undergoing electives surgery.
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Comparison of remifentanil and fentanyl in patients undergoing craniotomy for supratentorial space-occupying lesions.

TL;DR: This study compared the use of remifentanil or fentanyl during elective supratentorial craniotomy for space-occupying lesions and found that the former was more beneficial than the latter.
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Mild hypothermia as a protective therapy during intracranial aneurysm surgery: a randomized prospective pilot trial

TL;DR: The results indicate that a multicenter trial enrolling 300 to 900 patients with acute aneurysmal SAH will be required to demonstrate a statistically significant benefit with mild intraoperative hypothermia, feasible in nonobese patients and is well tolerated.
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Intracranial pressure and hemodynamic effects of remifentanil versus alfentanil in patients undergoing supratentorial craniotomy.

TL;DR: It is concluded that remifentanil produces similar cerebral perfusion pressure effects as does alfentanilic, and is associated with a dose-dependent decrease in MAP.