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Bryan A. Strange
Researcher at Technical University of Madrid
Publications - 86
Citations - 6842
Bryan A. Strange is an academic researcher from Technical University of Madrid. The author has contributed to research in topics: Hippocampal formation & Medicine. The author has an hindex of 30, co-authored 70 publications receiving 5873 citations. Previous affiliations of Bryan A. Strange include University College London & Foundation Center.
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Functional organization of the hippocampal longitudinal axis
TL;DR: Together, anatomical studies and electrophysiological recordings in rodents suggest a model in which functional long-axis gradients are superimposed on discrete functional domains, which provides a potential framework to explain and test the multiple functions ascribed to the hippocampus.
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Automatic and intentional brain responses during evaluation of trustworthiness of faces.
TL;DR: The findings extend a proposed model of social cognition by highlighting a functional dissociation between automatic engagement of amygdala versus intentional engagement of STS in social judgment.
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Encoding of emotional memories depends on amygdala and hippocampus and their interactions
TL;DR: Data indicate a reciprocal dependence between amygdala and hippocampus during the encoding of emotional memories in patients with variable degrees of left hippocampal and amygdala pathology who performed a verbal encoding task during functional magnetic resonance imaging.
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Segregating the Functions of Human Hippocampus
TL;DR: This work measures hippocampal responses to novelty, using functional MRI (fMRI), during an item-learning paradigm generated from an artificial grammar system, and demonstrates a left anterior hippocampal response to both types of novelty and adaptation of these responses with stimulus familiarity.
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Beta-adrenergic modulation of emotional memory-evoked human amygdala and hippocampal responses.
TL;DR: The results suggest that human emotional memory is associated with a beta-adrenergic-dependent modulation of amygdala-hippocampal interactions, and that administration of the propranolol at encoding abolishes the enhanced amygdala encoding and hippocampal retrieval effects.