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Byron H. Waksman
Researcher at Yale University
Publications - 93
Citations - 4909
Byron H. Waksman is an academic researcher from Yale University. The author has contributed to research in topics: Antigen & Spleen. The author has an hindex of 33, co-authored 93 publications receiving 4878 citations. Previous affiliations of Byron H. Waksman include Albert Einstein College of Medicine & Kyoto University.
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Potentiation of the t-lymphocyte response to mitogens : i. the responding cell
TL;DR: The importance of indirect effects in stimulating mitotic responses of T cells is emphasized by the fact that the mitotic response of mouse thymocytes to LPS and its ability to potentiate the response of these cells to PHA disappears with removal of adherent cells from the thymocyte population.
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Potentiation of the t-lymphocyte response to mitogens ii. the cellular source of potentiating mediator(s)
Igal Gery,Byron H. Waksman +1 more
TL;DR: It is concluded that LAF stimulates both central and peripheral T lymphocytes and enhances their responses to other stimulants.
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Cytotoxicity mediated by soluble antigen and lymphocytes in delayed hypersensitivity : iii. analysis of mechanism
Nancy H. Ruddle,Byron H. Waksman +1 more
TL;DR: The cytopathic effect of lymph node cells from tuberculin-sensitized rats on rat embryo fibroblasts in the presence of PPD was not enhanced by admixture of normal (nonsensitized) lymph nodes, and preincubation studies showed that this in vitro response is initiated by the reaction of lymphocytes with specific antigen.
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Cytotoxicity mediated by soluble antigen and lymphocytes in delayed hypersensitivity. I. Characterization of the phenomenon.
Nancy H. Ruddle,Byron H. Waksman +1 more
TL;DR: In the presence of specific antigen, lymph node cells from inbred rats with delayed hypersensitivity to tuberculoprotein, bovine gammaglobulin, and egg albumin produced progressive destruction of monolayers of rat embryo fibroblasts in tissue culture.
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On soluble mediators of immunologic regulation
Byron H. Waksman,Yuziro Namba +1 more