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C. Geiger-Kabisch

Publications -  17
Citations -  943

C. Geiger-Kabisch is an academic researcher. The author has contributed to research in topics: Dementia & Alzheimer's disease. The author has an hindex of 11, co-authored 17 publications receiving 909 citations.

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EEG coherence in Alzheimer disease

TL;DR: In this article, a novel approach is introduced to examine EEG coherence in different frequency bands of 17 locations from the 10-20 system. Fifty patients with clinically diagnosed Alzheimer's disease were compared with 42 age-approximated non-demented controls.
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Discrimination of Alzheimer's disease and normal aging by EEG data

TL;DR: This work studied 4 approaches to discriminate AD patients from controls by means of EEG data: Classification by group means, stepwise discriminant analysis, a neuronal network using back propagation and discriminantAnalysis preceded by principal components analysis (PCA).
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Parameters of EEG dimensional complexity in Alzheimer's disease

TL;DR: It is shown that patients with Alzheimer's disease have significantly lower dimensional complexity than age-approximated non-demented controls.
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Psychotic features and the course of Alzheimer's disease: relationship to cognitive, electroencephalographic and computerized tomography findings

TL;DR: Thirty‐one of 50 patients satisfying the NINCDS‐ADRDA criteria of probable or possible Alzheimer's disease showed psychotic features during a 2‐year observation period, which may indicate that specific psychotic features and their potential organic substrate exert an effect on the progression of illness and on survival in Alzheimer's Disease.
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Delusional misidentification in Alzheimer's disease: a summary of clinical and biological aspects

TL;DR: Patients with DMS showed increased EEG delta-power over the right hemisphere, their CT scans showed more severe right frontal lobe atrophy, and the number of their pyramidal cells in area CA1 was lower than in the patients without DMS, which may indicate that the development of DMS in AD can be promoted by certain patterns of brain degeneration which affect systems relevant to the recognition and updating of memories.