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Hans Förstl

Researcher at Technische Universität München

Publications -  391
Citations -  16596

Hans Förstl is an academic researcher from Technische Universität München. The author has contributed to research in topics: Dementia & Alzheimer's disease. The author has an hindex of 66, co-authored 388 publications receiving 15116 citations. Previous affiliations of Hans Förstl include Ludwig Maximilian University of Munich.

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Selective changes of resting-state networks in individuals at risk for Alzheimer's disease

TL;DR: This work analyzes functional and structural MRI data from healthy elderly and patients with amnestic mild cognitive impairment and concludes that in individuals at risk for AD, a specific subset of RSNs is altered, likely representing effects of ongoing early neurodegeneration.
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Analysis of Heterogeneous βA4 Peptides in Human Cerebrospinal Fluid and Blood by a Newly Developed Sensitive Western Blot Assay

TL;DR: It is found that CSF βA4 1-42 level is lower in AD patients compared with non-demented controls, although there was a significant overlap between the groups.
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Clinical features of Alzheimer's disease.

TL;DR: The life expectancy of patients with a clinical diagnosis of Alzheimer’s disease is significantly reduced, but to date there is hope that the period of relative well-being and not of suffering can be prolonged with modern symptomatic treatment interventions.
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Frontotemporal lobar degeneration: demographic characteristics of 353 patients.

TL;DR: These findings show that cohorts of patients can be combined using new research criteria for FTLD and demonstrate striking demographic differences among FTLD subgroups, including frontotemporal dementia, semantic dementia, and progressive nonfluent aphasia.
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Insular dysfunction within the salience network is associated with severity of symptoms and aberrant inter-network connectivity in major depressive disorder

TL;DR: Results provide evidence for a relationship between aberrant intra-iFC in the salience network's rAI, aberrant DMN/CEN interactions and severity of symptoms, suggesting a link between Aberrant salience mapping, abnormal coordination of DMN /CEN based cognitive processes and psychopathology in MDD.