C
Camila Hirotsu
Researcher at University Hospital of Lausanne
Publications - 94
Citations - 1926
Camila Hirotsu is an academic researcher from University Hospital of Lausanne. The author has contributed to research in topics: Polysomnography & Population. The author has an hindex of 19, co-authored 84 publications receiving 1405 citations. Previous affiliations of Camila Hirotsu include Federal University of São Paulo & University of Lausanne.
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Journal ArticleDOI
Interactions between sleep, stress, and metabolism: From physiological to pathological conditions
TL;DR: An overall view of the relationship between sleep, stress, and metabolism from basic physiology to pathological conditions is provided, highlighting effective treatments for metabolic disturbances.
Journal ArticleDOI
The NoSAS score for screening of sleep-disordered breathing: a derivation and validation study.
Helena Marti-Soler,Camila Hirotsu,Pedro Marques-Vidal,Peter Vollenweider,Gérard Waeber,Martin Preisig,Mehdi Tafti,Mehdi Tafti,Sergio Tufik,Lia Bittencourt,Sergio Tufik,José Haba-Rubio,Raphael Heinzer +12 more
TL;DR: The NoSAS score is a simple, efficient, and easy to implement score enabling identification of individuals at risk of sleep-disordered breathing and can help clinicians to decide which patients to further investigate with a nocturnal recording.
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Sleep fragmentation promotes NADPH oxidase 2-mediated adipose tissue inflammation leading to insulin resistance in mice
Shelley X. L. Zhang,Abdelnaby Khalyfa,Yang Wang,Alba Carreras,Fahed Hakim,Brian A. Neel,Matthew J. Brady,Zhuanhong Qiao,Camila Hirotsu,David Gozal +9 more
TL;DR: These studies imply that SF, a frequent occurrence in many disorders and more specifically in sleep apnea, is a potent inducer of insulin resistance via activation of oxidative stress and inflammatory pathways, thereby opening the way for therapeutic strategies.
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Sleep Loss and Cytokines Levels in an Experimental Model of Psoriasis
TL;DR: Data suggest that sleep deprivation plays an important role in the exacerbation of psoriasis through modulation of the immune system in the epidermal barrier, and sleep loss should be considered a risk factor for the development of psoriatic groups.
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Chronic sleep fragmentation during the sleep period induces hypothalamic endoplasmic reticulum stress and PTP1b-mediated leptin resistance in male mice.
TL;DR: In this paper, the effects of sleep fragmentation on the unfolded protein response (UPR) pathway and leptin receptor signaling in the hypothalamus were examined in wild-type mice treated with the ER chaperone tauroursodeoxycholic acid (TUDCA), as well as in CHOP-/- transgenic mice.