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Camille Martinand-Mari

Researcher at University of Montpellier

Publications -  23
Citations -  9967

Camille Martinand-Mari is an academic researcher from University of Montpellier. The author has contributed to research in topics: Programmed cell death & Gene. The author has an hindex of 12, co-authored 20 publications receiving 8608 citations.

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Cell death and renewal during prey capture and digestion in the carnivorous sponge Asbestopluma hypogea (Porifera: Poecilosclerida)

TL;DR: The results demonstrate that dynamics of cell renewal in sponge appear to be regulated by cellular mechanisms as multiple and complex as those already identified in bilaterian metazoans.
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Topological Control of Life and Death in Non-Proliferative Epithelia

TL;DR: Evidence is shown that massive apoptosis can also be controlled and coordinated by a pre-established pattern of a specific ‘master cell’ population, supported by the development and validation of an original model of cell patterning.
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Identification of autophagy genes in Ciona intestinalis: a new experimental model to study autophagy mechanism.

TL;DR: The genomes of Ciona intestinalis, of the second ascidian Ciona savignyi, and those of the classical biological models are explored to extract and compare autophagy gene sequences to conclude that a complex autophagic machinery is present in C Fiona intestinalis.
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Linking 2D observations to 3D modeling of enamel microstructure – a new integrative framework applied to hippopotamoidea evolutionary history

TL;DR: Simulenam as mentioned in this paper is a software dedicated to the simulation of enamel prisms, and an updated, integrative model of decussation formation is used to transform 2D observations of enamels into full 3D representations of hippopotamoid enamel microstructure.
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The non-proliferative nature of ascidian folliculogenesis as a model of highly ordered cellular topology distinct from proliferative epithelia.

TL;DR: A theoretical model of epithelial morphogenesis is developed, based on the physics of divided media, that takes into account biological parameters such as cell‐cell contact adhesions and tensions, cell and tissue growth, and which reproduces the effects of proliferation by increasing the topological heterogeneity observed experimentally.