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Charlotte Eitel

Researcher at University of Lübeck

Publications -  24
Citations -  584

Charlotte Eitel is an academic researcher from University of Lübeck. The author has contributed to research in topics: Myocardial infarction & Mace. The author has an hindex of 14, co-authored 24 publications receiving 406 citations.

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Prognostic Significance of Remote Myocardium Alterations Assessed by Quantitative Noncontrast T1 Mapping in ST-Segment Elevation Myocardial Infarction

TL;DR: In STEMI patients treated by PPCI, evaluation of remote zone alterations by quantitative noncontrast T1 mapping provided independent and incremental prognostic information in addition to clinical risk factors and traditional CMR outcome markers.
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Intramyocardial haemorrhage and prognosis after ST-elevation myocardial infarction.

TL;DR: IMH assessed by T2* imaging may provide prognostic information that is incremental to other CMR markers of infarct severity and classical clinical risk factors and could therefore be relevant as an important prognostic measure as well as therapeutic target when caring for patients after STEMI.
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Management of arrhythmias in patients with Takotsubo cardiomyopathy: Is the implantation of permanent devices necessary?

TL;DR: The data suggest that bradyarrhythmias in the acute setting of TTC may require permanent pacemaker implantation and, in contrast, polymorphic ventricular arrh rhythmias might be managed with a temporary approach until recovery of repolarization time and left ventricular function.
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Shock Index as a Predictor of Myocardial Damage and Clinical Outcome in ST-Elevation Myocardial Infarction.

TL;DR: STEMI patients with an elevated admission shock index had more pronounced myocardial and microvascular damage and the shock index was independently associated with MACE at 12 months and identified as an independent predictor of MACE.
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Optical Coherence Tomography to Evaluate Plaque Burden and Morphology in Patients With Takotsubo Syndrome.

TL;DR: In this article, the authors discuss the possibility of blocked atherosclerotic plaques in the left anterior descending coronary artery as a potential pathophysiological mechanism in Takotsubo syndrome (TTS).