C
Christina Bade-Doeding
Researcher at Hannover Medical School
Publications - 31
Citations - 467
Christina Bade-Doeding is an academic researcher from Hannover Medical School. The author has contributed to research in topics: Human leukocyte antigen & Peptide. The author has an hindex of 12, co-authored 31 publications receiving 408 citations.
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Journal ArticleDOI
HLA-E: A Novel Player for Histocompatibility
TL;DR: Structural investigations clearly demonstrate how subtle amino acid differences impact the strength and response of the cognate CD94/NKG2 or T cell receptor.
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Position 156 influences the peptide repertoire and tapasin dependency of human leukocyte antigen B*44 allotypes
Soumya Badrinath,Philippa Saunders,Trevor Huyton,Susanne Aufderbeck,Oliver Hiller,Rainer Blasczyk,Christina Bade-Doeding +6 more
TL;DR: The computational analysis suggests that the tapasin-independence of human leukocyte antigen B*44:28156Arg is a result of stabilization of the peptide binding region and generation of a more peptide receptive state, and the results of peptide sequencing suggest that tapasin chaperoning is needed to acquire peptides of unusual length.
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The molecular diversity of Sema7A, the semaphorin that carries the JMH blood group antigens
Axel Seltsam,Susanne Strigens,Cyril Levene,Vered Yahalom,Marilyn Moulds,John J. Moulds,H. Hustinx,Volker Weisbach,Dolores Figueroa,Christina Bade-Doeding,David S. DeLuca,Rainer Blasczyk +11 more
TL;DR: The present study was designed to elucidate the complex picture of the molecular diversity of Sema7A, the protein that carries the JMH blood group antigen, and plays an important role in axon growth and guidance.
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Weak blood group B phenotypes may be caused by variations in the CCAAT‐binding factor/NF‐Y enhancer region of the ABO gene
Axel Seltsam,Franz F. Wagner,Daniela Grüger,Christa Das Gupta,Christina Bade-Doeding,Rainer Blasczyk +5 more
TL;DR: The common ABO alleles were found to have CBF/NF‐Y enhancer regions with specific numbers of 43‐bp minisatellite repeats.
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A weak blood group A phenotype caused by a translation-initiator mutation in the ABO gene
TL;DR: Weak blood group A and B phenotypes are correlated with ABO glycosyltransferases exhibiting single‐amino‐acid changes and/or C‐terminal modifications.