C
Christopher R. Murphy
Researcher at University of Sydney
Publications - 194
Citations - 4188
Christopher R. Murphy is an academic researcher from University of Sydney. The author has contributed to research in topics: Blastocyst & Tight junction. The author has an hindex of 34, co-authored 188 publications receiving 3990 citations. Previous affiliations of Christopher R. Murphy include Stanford University & Flinders University.
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Uterine receptivity and the plasma membrane transformation
TL;DR: In this article, the authors examined the extensive alterations which occur in the plasma membrane of uterine epithelial cells during early pregnancy across species, and suggested that these changes are an ongoing process during much of early pregnancy, not just an event at the time attachment.
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Similar endometrial development in oocyte donors treated with either high- or standard-dose GnRH antagonist compared to treatment with a GnRH agonist or in natural cycles
Carlos Simón,J. Oberyé,José Bellver,C. Vidal,Ernesto Bosch,José A. Horcajadas,Christopher R. Murphy,Susan M. Adams,Anne Riesewijk,Bernadette Mannaerts,Antonio Pellicer +10 more
TL;DR: No relevant alteration was observed in the endometrial development in the early and mid-luteal phases in women undergoing controlled ovarian stimulation for oocyte donation following daily treatment with a standard- or high-dose GnRH antagonist.
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Interleukin-1 receptor antagonist prevents embryonic implantation by a direct effect on the endometrial epithelium
Carlos Simón,Diana Valbuena,Jan S. Krüssel,A. Bernal,Christopher R. Murphy,Tim Shaw,Antonio Pellicer,Mary Lake Polan +7 more
TL;DR: Impairment of embryonic adhesion with IL-1ra is mediated through a direct effect on transformation of the epithelial plasma membrane at the time of implantation as a result of down-regulation of alpha4, alpha v, and beta3.
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Estrogen Protects Lenses against Cataract Induced by Transforming Growth Factor-β (TGFβ)
TL;DR: It is shown that estrogen provides protection against cataract by countering the damaging effects of TGFβ, and adds to an increasing body of evidence that hormone replacement therapy protects postmenopausal women against various diseases.