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Chun Mao Lin

Researcher at Taipei Medical University

Publications -  69
Citations -  2213

Chun Mao Lin is an academic researcher from Taipei Medical University. The author has contributed to research in topics: Topoisomerase & Xanthine oxidase. The author has an hindex of 25, co-authored 68 publications receiving 1935 citations. Previous affiliations of Chun Mao Lin include Academia Sinica & Taipei Medical University Hospital.

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Assessment of renal function by the stable oxygen and hydrogen isotopes in human blood plasma.

TL;DR: The data indicate the existence of the biological homeostasis of water isotopes in all subjects, except the end stage renal disease subjects under the haemodialysis treatment, and the unexpected water contents in blood plasma of body water may shed light on a novel assessment of renal functions.
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Prevention of Cellular Oxidative Damage by an Aqueous Extract of Anoectochilus formosanus

TL;DR: The cell damage induced by oxidative stress was prevented by the plant extract in a concentration‐dependent manner, and the proteolytic cleavage of poly(ADP‐ribose) polymerase during the apoptotic process was also inhibited by AF extract.
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Uteroplacental Insufficiency Alters the Retinoid Pathway and Lung Development in Newborn Rats

TL;DR: The retinoid pathway may be one of the probable pathways mediating lung abnormalities caused by uteroplacental insufficiency, which causes defective alveolarization and transient increases in RAR-β expression in the lungs of newborn rats.
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Development of a novel latent electrochemical molecular substrate for the real-time monitoring of the tumor marker aminopeptidase N in live cells, whole blood and urine.

TL;DR: In this article , the ALA-AFC probe was used to detect Aminopeptidase N (APN/CD13) activity in a ratiometric electrochemical molecular substrate.
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C 60 fullerene nanoparticle prevents β-amyloid peptide induced cytotoxicity in neuro 2A cells

TL;DR: PEO-C60-3, a C60 fullerene derivative, was investigated against β-amyloid (Aβ)(subscript 25-35)-induced toxicity toward Neuro-2A cells in this study and offered new comprehension into the possible pathway of Aβ(subscript25-35) gene expression and C60 protective mechanism.