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Cornelia M. Wilson

Researcher at Canterbury Christ Church University

Publications -  40
Citations -  1708

Cornelia M. Wilson is an academic researcher from Canterbury Christ Church University. The author has contributed to research in topics: Lung cancer & Cancer. The author has an hindex of 17, co-authored 34 publications receiving 1322 citations. Previous affiliations of Cornelia M. Wilson include University of Liverpool & King Abdulaziz University.

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Tau protein kinases: involvement in Alzheimer's disease.

TL;DR: Tau protein kinases, their physiological roles and regulation, their involvement in tau phosphorylation and their relevance to AD are reviewed.
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Tau protein phosphatases in Alzheimer's disease: The leading role of PP2A

TL;DR: Tau protein phosphatases, their physiological roles and regulation, their involvement in tau phosphorylation and the relevance to AD are reviewed and the most common compounds acting on each t Tau phosphatase including PP2A are reviewed.
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Alternative Splicing and Protein Diversity: Plants Versus Animals.

TL;DR: A comprehensive summary of the current status of research in this area in both plants and humans is presented and the potential role of epigenetic modifications and chromatin state in splicing memory in plants primed with stresses is discussed.
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Eeyarestatin I inhibits Sec61-mediated protein translocation at the endoplasmic reticulum.

TL;DR: ESI acts by preventing the transfer of the nascent polypeptide from the co-translational targeting machinery to the Sec61 complex, which suggests that the drug can modulate canonical protein transport from the cytosol into the mammalian ER both in vitro and in vivo.
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Sortilin mediates the release and transfer of exosomes in concert with two tyrosine kinase receptors

TL;DR: It is demonstrated, in human lung cancer A549 cells, that the exosome release mechanism is closely linked to the multifaceted receptor sortilin (also called neurotensin receptor 3), and a new complex (termed the TES complex) is found in exosomes and results in the linkage of the two tyrosine kinase receptors TrkB and EGFR withsortilin.