D
Danièle Touati
Researcher at Centre national de la recherche scientifique
Publications - 32
Citations - 4101
Danièle Touati is an academic researcher from Centre national de la recherche scientifique. The author has contributed to research in topics: Superoxide dismutase & Superoxide. The author has an hindex of 25, co-authored 32 publications receiving 3922 citations. Previous affiliations of Danièle Touati include International Justice Mission.
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Journal ArticleDOI
Iron and oxidative stress in bacteria.
TL;DR: Iron metabolism, and its coupling with regulation of defenses against oxidative stress, as well as the role played by iron in regulatory protein in sensing redox change, appear as essential factors for life in the presence of oxygen.
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Lethal oxidative damage and mutagenesis are generated by iron in delta fur mutants of Escherichia coli: protective role of superoxide dismutase.
TL;DR: It is proposed that a transient iron overload occurs in wild-type cells on return to normal growth conditions following iron starvation, with the coupling between iron and MnSOD regulation helping the cells cope.
Journal ArticleDOI
Hfq, a new chaperoning role: binding to messenger RNA determines access for small RNA regulator.
Thomas Geissmann,Danièle Touati +1 more
TL;DR: Hfq is a critical chaperone in vivo and in vitro, changing the folding of the target mRNA to make it subject to the small RNA regulator.
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Oxidative burst in alfalfa-Sinorhizobium meliloti symbiotic interaction
TL;DR: It is shown that alfalfa responds to infection with Sinorhizobium meliloti by production of superoxide and hydrogen peroxide, which addresses the question of which mechanism rhizobia use to counteract the plant defense response.
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Oxygen-dependent mutagenesis in Escherichia coli lacking superoxide dismutase
TL;DR: The enhanced mutagenesis in aerobically grown sodA sodB mutants is largely dependent on functional exonuclease III, suggesting that the increased flux of superoxide radicals results in DNA lesions that can be acted on by this enzyme, leading to mutations.