D
Dennis P. Orgill
Researcher at Brigham and Women's Hospital
Publications - 452
Citations - 23889
Dennis P. Orgill is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Wound healing & Medicine. The author has an hindex of 70, co-authored 412 publications receiving 20440 citations. Previous affiliations of Dennis P. Orgill include University of Kentucky & University of Lausanne.
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Systematic review of coaching to enhance surgeons' operative performance
Hyeyoun Min,Dianali Rivera Morales,Dennis P. Orgill,Dennis P. Orgill,Douglas S. Smink,Steven Yule,Steven Yule +6 more
TL;DR: Surgical coaching interventions have a positive impact on learners' perception and attitudes, their technical and nontechnical skills, and performance measures and will benefit future coaching strategies and implementation to enhance operative performance.
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Reduction in incidence of deep sternal wound infections: random or real?
Evan Matros,Sary F. Aranki,Lauren R. Bayer,Siobhan McGurk,Jennifer Neuwalder,Dennis P. Orgill +5 more
TL;DR: Analysis of a large series of cardiac surgical patients demonstrates significant reduction in deep sternal wound infection incidence in 15 years, and introduction of perioperative intravenous insulin may explain some observed risk reduction.
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A protocol for the development of reporting criteria for surgical case reports: The SCARE statement
Riaz Agha,Alexander J. Fowler,Alexandra Saetta,Ishani Barai,Shivanchan Rajmohan,Dennis P. Orgill +5 more
TL;DR: The objective of this research is to conduct a Delphi consensus exercise amongst experienced case report reviewers and editors to develop the Surgical CAse REport (SCARE) Guidelines.
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Clinical applications of tissue engineered constructs.
TL;DR: The reconstruction of soft tissue defects poses a challenge for plastic surgeons and tissue engineers and the co-mingling of current reconstructive modalities with more aggressive and successful tissue engineering technology and the rapidly developing science of stem cell biology is likely to involve.
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Mature B cells accelerate wound healing after acute and chronic diabetic skin lesions.
Ruxandra F. Sîrbulescu,Chloe K. Boehm,Erin Soon,Moses Q. Wilks,Iulian Ilieş,Hushan Yuan,Ben Maxner,Nicolas A. Chronos,Charalambos Kaittanis,Marc D. Normandin,Georges El Fakhri,Dennis P. Orgill,Ann E. Sluder,Mark C. Poznansky +13 more
TL;DR: The findings indicate that the timeline and efficacy of wound healing can be experimentally manipulated through the direct application of mature, naive B cells, which effectively modify the balance of mature immune cell populations within the wound microenvironment and accelerate the healing process.