https://science.sciencemag.org/content/sci/5/110/206.full.pdf

The american psychological association.

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Research methods in the social sciences

In A General Theory of Crime, Gottfredson and Hirschi propose that low self-control, in interaction with criminal opportunity, is the major cause of crime. The research reported in this article attempts to test this argument while closely following the nominal definitions presented by Gottfredson and Hirschi. A factor analysis of items designed to measure low self-control is consistent with their contention that the trait is unidimensional. Further, the proposed interaction effect is found for self-reported acts of both fraud and force (their definition of crime). Inconsistent with the theory are (a) the finding that criminal opportunity has a significant main effect, beyond its interaction with low self-control, on self-reported crime and (b) the substantial proportion of variance in crime left unexplained by the theoretical variables. Suggestions are offered for modifying and expanding the theory.

Testing the Core Empirical Implications of Gottfredson and Hirschi's General Theory of Crime:

Here I argue that addicted people become unable to make drug-use choices on the basis of long-term outcome, and I propose a neural framework that explains this myopia for future consequences. I suggest that addiction is the product of an imbalance between two separate, but interacting, neural systems that control decision making: an impulsive, amygdala system for signaling pain or pleasure of immediate prospects, and a reflective, prefrontal cortex system for signaling pain or pleasure of future prospects. After an individual learns social rules, the reflective system controls the impulsive system via several mechanisms. However, this control is not absolute; hyperactivity within the impulsive system can override the reflective system. I propose that drugs can trigger bottom-up, involuntary signals originating from the amygdala that modulate, bias or even hijack the goal-driven cognitive resources that are needed for the normal operation of the reflective system and for exercising the willpower to resist drugs.

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Decision making, impulse control and loss of willpower to resist drugs: a neurocognitive perspective

This report builds on a highly valued predecessor, the 1994 Institute of Medicine (IOM) report entitled Reducing Risks for Mental Disorders: Frontiers for Preventive Intervention Research. That report provided the basis for understanding prevention science, elucidating its then-existing research base, and contemplating where it should go in the future. This report documents that an increasing number of mental, emotional, and behavioral problems in young people are in fact preventable. The proverbial ounce of prevention will indeed be worth a pound of cure: effectively applying the evidence-based prevention interventions at hand could potentially save billions of dollars in associated costs by avoiding or tempering these disorders in many individuals. Furthermore, devoting significantly greater resources to research on even more effective prevention and promotion efforts, and then reliably implementing the findings of such research, could substantially diminish the human and economic toll.

Preventing Mental, Emotional, and Behavioral Disorders Among Young People: Progress and Possibilities

Risky decision making and the anterior cingulate cortex in abstinent drug abusers and nonusers.

Impulsivity, aggression, and neuroendocrine responses to serotonergic stimulation in substance abusers

Delineation of the biological substrates of violence in substance abusers would be valuable in developing therapeutic interventions for substance abusers who tend to be refractory to conventional treatments. Substance abusers with chronically violent behavior show more severity and poorer prognosis of their substance abuse disorder and more criminal activity. Violence may share many of the same underlying mechanisms as substance abuse, given the comorbidity and commonalities in behavioral dimensions, including childhood and personality disorders with biological underpinnings. Neuropsychological dysfunction consistently characterizes both drug abuse and violence and may contribute to traits often cited as precursors to both, for example, impulsivity, poor decision-making ability, disinhibition, and inability to assess consequences. Deficits in executive cognitive functioning subserved specifically by the prefrontal cortex are implicated in substance abusers with violent behavior. Language: en

Neuropsychological Function, Drug Abuse, and Violence A Conceptual Framework

For several decades, mainstream criminology has been dominated by sociological and political perspectives. Although findings from these fields must not be discarded or underplayed, considered alone, they do not offer a complete assessment of the contributions to criminal behavior. Data currently being generated from numerous behavioral sciences, such as behavioral genetics, physiological psychology, psychopharmacology, and endocrinology, indicate that biological factors play an equally significant role in the development of antisocial behavior and should be considered accordingly. Incorporation of the theoretical parameters and findings of these behavioral sciences into a criminological framework would yield valuable in formation regarding processes underlying antisocial behavior. Such a multidisciplinary approach is likely to enhance capabilities to predict, prevent, and manage antisocial behavior. Theoretical parameters, methodological issues, selected research findings, potential applications, and precautions are discussed.

Diana Fishbein

Papers

Risky decision making and the anterior cingulate cortex in abstinent drug abusers and nonusers.

Impulsivity, aggression, and neuroendocrine responses to serotonergic stimulation in substance abusers

Neuropsychological Function, Drug Abuse, and Violence A Conceptual Framework

Biological perspectives in criminology

Neurocognitive characterizations of Russian heroin addicts without a significant history of other drug use.