D
Dirk C. G. Straver
Researcher at Utrecht University
Publications - 7
Citations - 289
Dirk C. G. Straver is an academic researcher from Utrecht University. The author has contributed to research in topics: Multifocal motor neuropathy & Mismatch negativity. The author has an hindex of 5, co-authored 6 publications receiving 255 citations.
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Journal ArticleDOI
Multifocal motor neuropathy: diagnosis, pathogenesis and treatment strategies
Lotte Vlam,W. Ludo van der Pol,Elisabeth A. Cats,Dirk C. G. Straver,Sanneke Piepers,Hessel Franssen,Leonard H. van den Berg +6 more
TL;DR: The diagnostic criteria for MMN is discussed, available treatments and promising new therapeutic strategies are described, and an update on the current understanding of MMN pathogenesis is provided.
Journal ArticleDOI
Pathophysiology of immune-mediated demyelinating neuropathies--Part II: Neurology.
TL;DR: The relationship between pathophysiology and symptoms and thepathophysiology of specific disease entities are described and discussed, including Guillain–Barré syndrome, chronic inflammatory demyelinating polyneuropathy, multifocal motor neuropathy, anti–myelin‐associated glycoprotein neuropathies, and POEMS syndrome.
Journal ArticleDOI
MMN: From Immunological Cross-Talk to Conduction Block
Oliver Harschnitz,Bas A. Jongbloed,Hessel Franssen,Dirk C. G. Straver,W. Ludo van der Pol,Leonard H. van den Berg +5 more
TL;DR: The current understanding of the immune pathogenesis underlying MMN and how this may cause CB, available treatment strategies and future therapeutic targets are discussed.
Journal ArticleDOI
Cold paresis in multifocal motor neuropathy
Dirk C. G. Straver,Jan-Thies H. van Asseldonk,Nicolette C. Notermans,John H. J. Wokke,Leonard H. van den Berg,Hessel Franssen +5 more
TL;DR: Symptoms of cold paresis are common in peripheral nervous system disorders, particularly in MMN, and this supports the hypothesis that cold plesis inMMN does not reflect demyelination only, but may indicate the existence of inflammatory nerve lesions with permanently depolarized axons that only just conduct at normal temperature, but fail at lower temperatures.
Journal ArticleDOI
Spinal arteriovenous shunts presenting as intracranial subarachnoid haemorrhage
TL;DR: Investigation of the characteristics of patients with SAVS who present with intracranial SAH without symptoms and signs suggesting a spinal cause found one patient (0.05%, 95 % CI 0.006–0.3%).