E
Edward G. Pasanen
Researcher at University of Texas at Austin
Publications - 44
Citations - 1316
Edward G. Pasanen is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Otoacoustic emission & Poison control. The author has an hindex of 20, co-authored 44 publications receiving 1265 citations.
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Lateralization at high frequencies based on interaural time differences
TL;DR: In many conditions of listening, sensitivity to interaural time differences at high frequencies compares favorably with sensitivity at low frequencies—good performace requires only tens of microseconds of interaurally time delay.
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Comparison of the auditory systems of heterosexuals and homosexuals: Click-evoked otoacoustic emissions
TL;DR: The CEOAEs of homosexual and bisexual females were found to be intermediate to those of heterosexual females and heterosexual males, which is a parsimonious explanation for the change in their sexual orientation.
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Partial dissociation of spontaneous otoacoustic emissions and distortion products during aspirin use in humans
TL;DR: Aspirin consumption reduced the amplitude of the evoked distortion products (EDPs) but did not eliminate them entirely, and at low primary levels, EDPs near the SOAE frequency were 10-20 dB higher than when they were 100 Hz away from theSOAE frequency.
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Aspirin-induced hearing loss as a model of sensorineural hearing loss.
TL;DR: In this paper, the authors used a model condition for sensorineural hearing loss, where the hearing loss is not asymmetrically distributed toward the high frequency region, as it typically is with mild sensorinaural deafness, and large individual differences in the amount of temporary hearing loss induced by fixed doses of aspirin.
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Spontaneous otoacoustic emissions in heterosexuals, homosexuals, and bisexuals.
TL;DR: The SOAE and CEOAE data both suggest that the cochleas of homosexual and bisexual females have been partially masculinized, possibly as part of some prenatal processes that also masculinizes whatever brain structures are responsible for sexual orientation.