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Edward L. Yellin
Researcher at Albert Einstein College of Medicine
Publications - 67
Citations - 2966
Edward L. Yellin is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Mitral valve & Diastole. The author has an hindex of 25, co-authored 66 publications receiving 2927 citations.
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Journal ArticleDOI
Left ventricular filling dynamics: influence of left ventricular relaxation and left atrial pressure.
Y. Ishida,Jay S. Meisner,K. Tsujioka,J. I. Gallo,C Yoran,Robert W.M. Frater,Edward L. Yellin +6 more
TL;DR: PRFR is determined by both the left atrial pressure and the left ventricular relaxation rate and should be used with caution as an index ofleft ventricular diastolic function.
Journal ArticleDOI
Passive properties of canine left ventricle: diastolic stiffness and restoring forces.
Srdjan D. Nikolic,Edward L. Yellin,Koichi Tamura,H. O. Vetter,T. Tamura,Jay S. Meisner,Robert W.M. Frater +6 more
TL;DR: In this paper, the authors proposed a logarithmic approach to quantify the passive properties of the intact ventricle and the effects of elastic recoil by separating filling from relaxation with a method of volume clamping with a remote-controlled mitral valve.
Journal ArticleDOI
Left ventricular relaxation in the filling and nonfilling intact canine heart
Edward L. Yellin,Masatsugu Hori,Chaim Yoran,Edmund H. Sonnenblick,S. Gabbay,Robert W.M. Frater +5 more
TL;DR: Left ventricular relaxation in the filling and transiently nonfilling working hearts of seven open-chest pentobarbital-anesthetized dogs is studied by totally occluding the mitral annulus during one systole to evaluate the validity of a monoexponential characterization of relaxation.
Journal ArticleDOI
Left ventricular filling dynamics and diastolic function.
Journal ArticleDOI
Dynamic aspects of acute mitral regurgitation: effects of ventricular volume, pressure and contractility on the effective regurgitant orifice area.
TL;DR: Findings support the clinical view that maintaining a small LV with sustained myocardial contractility will reduce mitral regurgitation and left ventricular dilatation can enhance mitral Regurgitation by increasing the effective regurgitant orifice independent of SPG.