E
Elise G. Lavoie
Researcher at University of Arkansas for Medical Sciences
Publications - 37
Citations - 1675
Elise G. Lavoie is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Hepatic stellate cell & Extracellular. The author has an hindex of 20, co-authored 36 publications receiving 1530 citations. Previous affiliations of Elise G. Lavoie include University of Arkansas & Laval University.
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Journal ArticleDOI
Comparative hydrolysis of P2 receptor agonists by NTPDases 1, 2, 3 and 8.
Filip Kukulski,Sébastien A. Lévesque,Elise G. Lavoie,Joanna Lecka,François Bigonnesse,Aileen F. Knowles,Simon C. Robson,Terence L. Kirley,Jean Sévigny +8 more
TL;DR: Data indicate that plasma membrane NTPDases hydrolyze nucleotides in a distinctive manner and may therefore differentially regulate P2 and adenosine receptor signaling.
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Nucleoside Triphosphate diphosphohydrolase-2 Is the ecto-ATPase of Type I Cells in Taste Buds
TL;DR: Analysis of the double‐labeled sections indicates that NTPDase2 immunoreactivity is found on cell processes that often envelop other taste cells, reminiscent of type I cells, indicating that the presence of ecto‐ATPase in taste buds likely reflects the importance of ATP as an intercellular signaling molecule in this system.
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Specificity of the ecto‐ATPase inhibitor ARL 67156 on human and mouse ectonucleotidases
TL;DR: This work has identified the ectonucleotidases inhibited by ARL 67156 and described their role in the hydrolysis of ATP at the cell surface.
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Portal fibroblasts regulate the proliferation of bile duct epithelia via expression of NTPDase2
TL;DR: In this paper, portal fibroblasts were found to regulate bile duct epithelial P2Y receptors via expression of the ecto-nucleotidase NTPDase2.
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P2Y2 Receptor Transcription Is Increased by NF-κB and Stimulates Cyclooxygenase-2 Expression and PGE2 Released by Intestinal Epithelial Cells
Émilie Degagné,Djordje Grbic,Andrée-Anne Dupuis,Elise G. Lavoie,Christine Langlois,Nishant Jain,Gary A. Weisman,Jean Sévigny,Fernand-Pierre Gendron +8 more
TL;DR: It is demonstrated that P2Y2R expression is regulated during intestinal inflammation through an NF-κB p65-dependent mechanism and could contribute not only to inflammatory bowel disease but also to other inflammatory diseases by regulating PG release.