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Erik A. Visser

Researcher at University of Pretoria

Publications -  11
Citations -  205

Erik A. Visser is an academic researcher from University of Pretoria. The author has contributed to research in topics: Pinus patula & Fusarium circinatum. The author has an hindex of 6, co-authored 8 publications receiving 129 citations.

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Combined de novo and genome guided assembly and annotation of the Pinus patula juvenile shoot transcriptome

TL;DR: The assembled transcriptome will serve as a major genomic resource for future investigation of P. patula and represents the largest gene catalogue produced to date for this species, and could be applied to generate similar resources for other non-model species.
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Uncovering the defence responses of Eucalyptus to pests and pathogens in the genomics age.

TL;DR: The survey of PR genes in Eucalyptus provides a first step in identifying defence gene targets that may be employed for protection of the species in future and synthesizes existing knowledge of defence mechanisms in model plants and tree species.
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Dual RNA-Sequencing to Elucidate the Plant-Pathogen Duel.

TL;DR: This review focuses on the design of dual RNA-seq experiments and the application of downstream data analysis to gain biological insight into both sides of the interaction and a reduction in sequencing cost and single cell transcriptomics coupled with protein and metabolite level dual approaches are set to enhance understanding of plant-pathogen interactions.
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Defence transcriptome assembly and pathogenesis related gene family analysis in Pinus tecunumanii (low elevation).

TL;DR: The P. tecunumanii transcriptome is the first gene catalogue for the species, representing an important resource for studying resistance to the pitch canker pathogen, F. circinatum.
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Dual RNA-Seq Analysis of the Pine-Fusarium circinatum Interaction in Resistant (Pinus tecunumanii) and Susceptible (Pinus patula) Hosts.

TL;DR: It is suggested that coordination of phytohormone signaling is required for F. circinatum resistance in P. tecunumanii, while a comparatively delayed response and impaired phytosorbent signaling contributes to susceptibility in P P. patula.