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Eugenia Russinova

Researcher at Ghent University

Publications -  115
Citations -  7839

Eugenia Russinova is an academic researcher from Ghent University. The author has contributed to research in topics: Arabidopsis & Brassinosteroid. The author has an hindex of 41, co-authored 93 publications receiving 6255 citations. Previous affiliations of Eugenia Russinova include De Montfort University & University of Castilla–La Mancha.

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Heterodimerization and endocytosis of Arabidopsis brassinosteroid receptors BRI1 and AtSERK3 (BAK1).

TL;DR: It is proposed that the AtSERK3 protein is involved in changing the equilibrium between plasma membrane–located BRI1 homodimers and endocytosed B RI1-At SERK3 heterodimmers.
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Brassinosteroids: Multidimensional Regulators of Plant Growth, Development, and Stress Responses.

TL;DR: Recent progress toward understanding theBR pathway is summarized, including BR perception and the molecular mechanisms of BR signaling, and how knowledge of the BR pathway is being applied to manipulate the growth and stress responses of crops is shown.
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Brassinosteroids control meristem size by promoting cell cycle progression in Arabidopsis roots

TL;DR: Evidence is provided that BRs play a regulatory role in the control of cell-cycle progression and differentiation in the Arabidopsis root meristem and in the stem cell niche, BRs promote the QC renewal and differentiation of distal stem cells.
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The Arabidopsis thaliana SOMATIC EMBRYOGENESIS RECEPTOR-LIKE KINASES1 and 2 Control Male Sporogenesis

TL;DR: The Arabidopsis thaliana SOMATIC EMBRYOGENESIS RECEPTOR-LIKE KINASE (SERK) family of plasma membrane receptors consists of five closely related members and the SERK1 and SERK2 genes show a complex expression pattern throughout development, suggesting they are interchangeable in theSERK1/SERK2 signaling complex.
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The Arabidopsis SOMATIC EMBRYOGENESIS RECEPTOR-LIKE KINASE1 protein complex includes BRASSINOSTEROID-INSENSITIVE1

TL;DR: Results suggest that apart from SERK3, SERK1 is also involved in the brassinolide signaling pathway.