scispace - formally typeset
F

Fabrice Gankam Kengne

Researcher at Université libre de Bruxelles

Publications -  12
Citations -  503

Fabrice Gankam Kengne is an academic researcher from Université libre de Bruxelles. The author has contributed to research in topics: Hyponatremia & Hypertonic saline. The author has an hindex of 7, co-authored 12 publications receiving 401 citations. Previous affiliations of Fabrice Gankam Kengne include University of Texas Southwestern Medical Center & Erasmus University Rotterdam.

Papers
More filters
Journal ArticleDOI

Treatment of euvolemic hyponatremia in the intensive care unit by urea

Guy Decaux, +3 more
- 14 Oct 2010 - 
TL;DR: Data show that urea is a simple and inexpensive therapy to treat euvolemic hyponatremia in the ICU, and all the patients with neurological symptoms made a rapid recovery.
Journal ArticleDOI

Re-induction of hyponatremia after rapid overcorrection of hyponatremia reduces mortality in rats

Fabrice Gankam Kengne, +5 more
- 02 Sep 2009 - 
TL;DR: The results suggest that after inadvertent rapid correction of hyponatremia, treatment options should favor re-lowering serum sodium, and the increased permeability of blood-brain barrier seen in osmotic demyelination syndrome may not be a primary pathophysiologic insult of this syndrome.
Journal ArticleDOI

Astrocytes Are an Early Target in Osmotic Demyelination Syndrome

Fabrice Gankam Kengne, +7 more
- 01 Oct 2011 - 
TL;DR: A rat model of osmotic demyelination syndrome found that massive astrocyte death occurred after rapid correction of hyponatremia, delineating the regions of future myelin loss and supporting a model for the pathophysiology of Osmotic brain injury.
Journal ArticleDOI

Hyponatremia and the Brain.

Fabrice Gankam Kengne, +1 more
- 01 Jan 2018 - 
TL;DR: The brain can be affected by the treatment of hyponatremia, which, if not undertaken cautiously, could lead to osmotic demyelination syndrome, a rare demYelinating brain disorder that occurs after rapid correction of severe hypon atremia.
Journal ArticleDOI

In vivo morphological changes in animal models of amyotrophic lateral sclerosis and Alzheimer's-like disease: MRI approach.

Pavle R. Andjus, +12 more
- 01 Dec 2009 - 
TL;DR: In both models, Gd‐DTPA contrast revealed a compromised blood brain barrier that may serve as the passage for inflammatory immune cells in the vicinity of dilated lateral ventricles, thus revealing a potentially vulnerable point that can be the primary target of neurodegeneration in the central nervous system.