F
Fahad Khan
Researcher at Noida Institute of Engineering and Technology
Publications - 44
Citations - 513
Fahad Khan is an academic researcher from Noida Institute of Engineering and Technology. The author has contributed to research in topics: Apoptosis & Cell cycle. The author has an hindex of 9, co-authored 39 publications receiving 219 citations. Previous affiliations of Fahad Khan include Integral University.
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Journal ArticleDOI
Andrographolide Exhibits Anticancer Potential Against Human Colon Cancer Cells by Inducing Cell Cycle Arrest and Programmed Cell Death via Augmentation of Intracellular Reactive Oxygen Species Level.
TL;DR: The results indicated that andrographolide exhibited antiproliferative and apoptotic properties against colon cancer HT-29 cells.
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Glycyrrhizin induces reactive oxygen species-dependent apoptosis and cell cycle arrest at G0/G1 in HPV18+ human cervical cancer HeLa cell line.
TL;DR: Glycyrrhizin exposure significantly reduced the cell viability of HeLa cells with a concomitant increase in nuclear condensation and DNA fragmentation in a dose dependent manner and induced apoptosis in cervical cancer cells by exerting mitochondrial depolarization.
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Carvacrol Induces Reactive Oxygen Species (ROS)-mediated Apoptosis Along with Cell Cycle Arrest at G 0/G 1 in Human Prostate Cancer Cells
TL;DR: It is established that carvacrol could be a promising chemotherapeutic agent and could have a direct practical implication and translational relevance to prostate cancer patients as Origanum consumption may retard prostate cancer progression.
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A mechanistic review of the anticancer potential of hesperidin, a natural flavonoid from citrus fruits.
Pratibha Pandey,Fahad Khan +1 more
TL;DR: Hesperidin, a phytoactive compound, is an abundant and economical dietary bioflavonoid possessing numerous biological and medicinal benefits as discussed by the authors, which has been reported to alter several molecular targets related to carcinogenesis, such as reactive nitrogen species, cellular kinases, transcription factors, reactive oxygen species, drug transporters, cell cycle mediators and inflammatory cytokines.
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Carvacrol Induced Program Cell Death and Cell Cycle Arrest in Androgen-Independent Human Prostate Cancer Cells via Inhibition of Notch Signaling.
TL;DR: The results suggested that the carvacrol treatment significantly reduced the cell viability of PC-3 cells in a dose- and time-dependent manner and established the strong potential of carVacrol as a chemopreventive agent against androgen-independent human prostate cancer cells.