F
Faiz Ul Amin
Researcher at UPRRP College of Natural Sciences
Publications - 17
Citations - 1269
Faiz Ul Amin is an academic researcher from UPRRP College of Natural Sciences. The author has contributed to research in topics: Neuroprotection & Oxidative stress. The author has an hindex of 13, co-authored 17 publications receiving 824 citations. Previous affiliations of Faiz Ul Amin include Gyeongsang National University & University of Agriculture, Peshawar.
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Journal ArticleDOI
Natural Dietary Supplementation of Anthocyanins via PI3K/Akt/Nrf2/HO-1 Pathways Mitigate Oxidative Stress, Neurodegeneration, and Memory Impairment in a Mouse Model of Alzheimer's Disease.
Tahir Ali,Tae Hyun Kim,Shafiq Ur Rehman,Muhammad Sohail Khan,Faiz Ul Amin,Mehtab Khan,Muhammad Ikram,Myeong Ok Kim +7 more
TL;DR: It is suggested that consumption and supplementation of natural-derived anti-oxidant neuroprotective agent such as anthocyanins may be beneficial and suggest new dietary-supplement strategies for intervention in and prevention of progressive neurodegenerative diseases, such as AD.
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Melatonin Stimulates the SIRT1/Nrf2 Signaling Pathway Counteracting Lipopolysaccharide (LPS)-Induced Oxidative Stress to Rescue Postnatal Rat Brain.
TL;DR: The first detailed description of the mechanism of melatonin neuroprotection against LPS‐induced oxidative stress, acute neuroinflammation, and neurodegeneration in the hippocampal dentate gyrus (DG) region of the postnatal day 7 (PND7) rat brain is provided.
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Vanillic acid attenuates Aβ 1-42 -induced oxidative stress and cognitive impairment in mice
TL;DR: This study is the first to reveal the neuroprotective effect of VA against Aβ1-42-induced neurotoxicity and demonstrates that VA could potentially serve as a novel, promising, and accessible neuroprot protective agent against progressive neurodegenerative diseases such as AD.
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Anthocyanins encapsulated by PLGA@PEG nanoparticles potentially improved its free radical scavenging capabilities via p38/JNK pathway against Aβ1-42-induced oxidative stress.
TL;DR: The first time anthocyanins were encapsulated in biodegradable nanoparticle formulation based on poly (lactide-co-glycolide) (PLGA) and a stabilizer polyethylene glycol (PEG)-2000 and this data confirmed the therapeutic potential of Anthocyanin loaded nanoparticles in reducing AD pathology and offer an effective way to improve the efficiency of anthcyanins through the use of nanodrug delivery systems.
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Enhanced neuroprotection of anthocyanin-loaded PEG-gold nanoparticles against Aβ1-42-induced neuroinflammation and neurodegeneration via the NF-KB /JNK/GSK3β signaling pathway.
TL;DR: Results demonstrate that PEG-coated gold anthocyanins nanoparticles could be a new therapeutic agent in the field of nanomedicine to prevent neurodegenerative diseases such as AD.