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Farina Hanif

Researcher at Dow University of Health Sciences

Publications -  19
Citations -  760

Farina Hanif is an academic researcher from Dow University of Health Sciences. The author has contributed to research in topics: Medicine & Temozolomide. The author has an hindex of 6, co-authored 13 publications receiving 451 citations. Previous affiliations of Farina Hanif include University of Karachi.

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Glioblastoma Multiforme: A Review of its Epidemiology and Pathogenesis through Clinical Presentation and Treatment

TL;DR: It was found that radiation and certain genetic syndromes are the only risk factors identified to date for GBM, and the pathogenesis to involve aberrations of multiple signaling pathways through multiple genetic mutations and altered gene expression.
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Modulation of c-Fos and BDNF protein expression in pentylenetetrazole-kindled mice following the treatment with novel antiepileptic compound HHL-6.

TL;DR: One of the possible mechanisms of HHL-6 to inhibit epileptogenesis might be due to its controlling effect on the cellular and molecular expression of the factors that contribute to the development of epileptogenic plasticity in the CNS.
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N-(2-hydroxyphenyl)acetamide (NA-2) and Temozolomide synergistically induce apoptosis in human glioblastoma cell line U87

TL;DR: Findings support that NA-2 possesses strong apoptotic activity and the combined administration of NA- 2 and TMZ may be therapeutically exploited for the management of GBM.
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N-(2-hydroxy phenyl) acetamide: a novel suppressor of Toll-like receptors (TLR-2 and TLR-4) in adjuvant-induced arthritic rats

TL;DR: It is suggested that NA-2 affects AIA in a pleiotropic manner, suppressing TLRs-mediated joint inflammation and related symptoms, and is associated with improved histology.
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Relation of Copeptin with Diabetic and Renal Function Markers Among Patients with Diabetes Mellitus Progressing Towards Diabetic Nephropathy

TL;DR: The significant correlation of copeptin with diabetic and renal biomarkers, along with its positive association with family history of DM support its' role as an early and reliable biomarker of DM and its associated nephropathy.