F
Felix C. Tanner
Researcher at University of Zurich
Publications - 217
Citations - 9002
Felix C. Tanner is an academic researcher from University of Zurich. The author has contributed to research in topics: Tissue factor & Internal medicine. The author has an hindex of 46, co-authored 193 publications receiving 7884 citations. Previous affiliations of Felix C. Tanner include University of Pennsylvania.
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Journal ArticleDOI
Drug-Eluting Stent and Coronary Thrombosis: Biological Mechanisms and Clinical Implications
Thomas F. Lüscher,Jan Steffel,Franz R. Eberli,Michael Joner,Gaku Nakazawa,Felix C. Tanner,Renu Virmani +6 more
TL;DR: Evidence of a substantially higher risk of stent thrombosis in drug-eluting stents in DES remains inconclusive; as a consequence, both large-scale and long-term clinical trials are needed.
Journal ArticleDOI
Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease
Christian Besler,Kathrin Heinrich,Lucia Rohrer,Carola Doerries,Meliana Riwanto,Diana M. Shih,Angeliki Chroni,Keiko Yonekawa,Sokrates Stein,Nicola Schaefer,Maja Mueller,Alexander Akhmedov,Georgios Daniil,Costantina Manes,Christian Templin,Christophe Wyss,Willibald Maier,Felix C. Tanner,Christian M. Matter,Roberto Corti,Clement E. Furlong,Aldons J. Lusis,Arnold von Eckardstein,Alan M. Fogelman,Thomas F. Lüscher,Ulf Landmesser +25 more
TL;DR: Reduced HDL-associated paraoxonase 1 (PON1) activity is identified as one molecular mechanism leading to the generation of HDL with endothelial PKCβII-activating properties, at least in part due to increased formation of malondialdehyde in HDL.
Journal ArticleDOI
Tissue factor in cardiovascular diseases: molecular mechanisms and clinical implications.
TL;DR: Tissue Factor (TF) is the key initiator of the coagulation cascade; it binds factor VIIa resulting in activation of factor IX and factor X, ultimately leading to fibrin formation as mentioned in this paper.
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Inflammatory Markers at the Site of Ruptured Plaque in Acute Myocardial Infarction Locally Increased Interleukin-6 and Serum Amyloid A but Decreased C-Reactive Protein
Willibald Maier,Lukas Altwegg,Roberto Corti,Martin Hersberger,Friedrich E. Maly,Gabor Sütsch,Marco Roffi,Michel Neidhart,Franz R. Eberli,Felix C. Tanner,Sharon Gobbi,Arnold von Eckardstein,Thomas F. Lüscher +12 more
TL;DR: Coronary levels of IL-6 and SAA at the site of plaque rupture were increased relative to the systemic circulation, indicating local production of biologically active inflammatory mediators.
Journal ArticleDOI
Outcomes After Current Transcatheter Tricuspid Valve Intervention: Mid-Term Results From the International TriValve Registry
Maurizio Taramasso,Hannes Alessandrini,Azeem Latib,Masahiko Asami,Adrian Attinger-Toller,Luigi Biasco,Daniel Braun,Eric Brochet,Kim A. Connelly,Paolo Denti,Florian Deuschl,Andrea Englmeier,Neil Fam,Christian Frerker,Jörg Hausleiter,Dominique Himbert,Edwin C. Ho,Jean-Michel Juliard,Ryan Kaple,Felix Kreidel,Karl-Heinz Kuck,Marco Ancona,Alexander Lauten,Philipp Lurz,Michael Mehr,Tamin Nazif,Georg Nickening,Giovanni Pedrazzini,Alberto Pozzoli,Fabien Praz,Rishi Puri,Josep Rodés-Cabau,Ulrich Schäfer,Joachim Schofer,Horst Sievert,Kolja Sievert,Gilbert H.L. Tang,Felix C. Tanner,Alec Vahanian,John G. Webb,Stephan Windecker,Ermela Yzeiray,Michel Zuber,Francesco Maisano,Martin B. Leon,Rebecca T. Hahn +45 more
TL;DR: TTVI is feasible with different technologies, has a reasonable overall procedural success rate, and is associated with low mortality and significant clinical improvement, and mid-term survival is favorable in this high-risk population.