F
Frans J. Walther
Researcher at University of California, Los Angeles
Publications - 318
Citations - 10606
Frans J. Walther is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Pulmonary surfactant & Bronchopulmonary dysplasia. The author has an hindex of 53, co-authored 313 publications receiving 9719 citations. Previous affiliations of Frans J. Walther include Drew University & Leiden University.
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Journal ArticleDOI
Twin anemia-polycythemia sequence: diagnostic criteria, classification, perinatal management and outcome.
Femke Slaghekke,W. J. Kist,Dick Oepkes,S.A. Pasman,Johanna M. Middeldorp,Frans J.C.M. Klumper,Frans J. Walther,Frank P.H.A. Vandenbussche,Enrico Lopriore +8 more
TL;DR: This review focuses on the pathogenesis, incidence, diagnostic criteria, management options and outcome in twin-twin transfusion syndrome (TAPS), and proposes a classification system for antenatal and postnatal TAPS.
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A Randomized, Controlled Trial of Delivery-Room Respiratory Management in Very Preterm Infants
Arjan B. te Pas,Frans J. Walther +1 more
TL;DR: In the early functional residual capacity intervention group, fewer infants were intubated at <72 hours of age or received >1 dose of surfactant, and the average duration of ventilatory support was less, and Infants in the earlyfunctional residual Capacity intervention group developed bronchopulmonary dysplasia less frequently.
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Twin Anemia–Polycythemia Sequence in Two Monochorionic Twin Pairs Without Oligo-Polyhydramnios Sequence
Enrico Lopriore,Johanna M. Middeldorp,Dick Oepkes,Humphrey H.H. Kanhai,Frans J. Walther,Frank P.H.A. Vandenbussche +5 more
TL;DR: An uncommon form of chronic inter-twin transfusion, named TAPS, with severe anemia in one twin and polycythemia in the other, without the characteristically associated twin oligo-polyhydramnios sequence (TOPS), is reported, resulting in severe fetal or neonatal hematological complications.
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Gene expression profile and histopathology of experimental bronchopulmonary dysplasia induced by prolonged oxidative stress.
Gerry T. M. Wagenaar,Simone A. J. ter Horst,Margot A. Van Gastelen,Lara M. Leijser,Thais Mauad,Pieter A. van der Velden,Emile de Heer,Pieter S. Hiemstra,Ben J. H. M. Poorthuis,Frans J. Walther,Frans J. Walther +10 more
TL;DR: Differential gene expression with DNA microarray analysis in premature rat lungs exposed to prolonged hyperoxia during the saccular stage of development closely resembles the development of the lungs of premature infants receiving neonatal intensive care, and the magnitude of fibrin deposition is exciting.
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Looking back in time: outcome of a national cohort of very preterm infants born in The Netherlands in 1983
TL;DR: Although 90% of the children are without severe disabilities at school age, many of them meet serious difficulties in everyday life and the burden of mild developmental abnormalities, behavioral and learning disorders increases with age.