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Gaetano Magro

Researcher at University of Catania

Publications -  302
Citations -  5866

Gaetano Magro is an academic researcher from University of Catania. The author has contributed to research in topics: Myofibroblastoma & Medicine. The author has an hindex of 37, co-authored 282 publications receiving 5160 citations. Previous affiliations of Gaetano Magro include Casa Sollievo della Sofferenza.

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Spindle cell lipoma-like tumor, solitary fibrous tumor and myofibroblastoma of the breast: a clinico-pathological analysis of 13 cases in favor of a unifying histogenetic concept

TL;DR: It is postulate that the vimentin+/CD34+ cells of the mammary stroma, the well-known inherent plasticity of which to differentiate toward several mesenchymal lines, provides the explanation for the phenotypic heterogeneity of these neoplasms.
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Mammary myofibroblastoma: a tumor with a wide morphologic spectrum.

TL;DR: The incidence of MFB diagnosis has increased in recent years, likely due to the mammographic screening, and this unusual benign tumor may represent a potential diagnostic pitfall, especially when interpreting fine-needle aspiration and/or needle core biopsy.
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Solitary fibrous tumor of the central nervous system: a 15-year literature survey of 220 cases (August 1996-July 2011).

TL;DR: The anatomical distribution of the 220 cases reported so far reveals that most are intracranial and just over one-fifth are intraspinal, and preoperative imaging and intraoperative findings suggest meningioma, schwannoma or neurofibroma, hemangiopericytoma, or pituitary tumors.
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Protection from experimental autoimmune diabetes in the non‐obese diabetic mouse with soluble interleukin‐1 receptor

TL;DR: The anti‐diabetogenic property of sIL‐1R may not involve major T cell function impairment; accordingly, in parallel experiments, splenic lymphoid cells from NOD mice not challenged with CY, but treated with 2 mg/kg sIL-1R for 5 consecutive days showed a normal distribution of mononuclear cell subsets and maintained their capacity to secrete interferon‐γ and IL‐2 and to proliferate in response to polyclonal mitogenic stimulation with