G
Gail E. Christie
Researcher at Virginia Commonwealth University
Publications - 41
Citations - 1596
Gail E. Christie is an academic researcher from Virginia Commonwealth University. The author has contributed to research in topics: Gene & Bacteriophage P2. The author has an hindex of 23, co-authored 41 publications receiving 1582 citations. Previous affiliations of Gail E. Christie include University of California, Berkeley & University of Oregon.
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Journal ArticleDOI
The complete nucleotide sequence of the tryptophan operon of Escherichia coli
Charles Yanofsky,Terry Platt,Irving P. Crawford,Brian P. Nichols,Gail E. Christie,Heidi Horowitz,M. VanCleemput,Anna M. Wu +7 more
TL;DR: The complete nucleotide sequence for the five structural genes of the trp operon of E. coli together with the internal and flanking regions of regulatory information are presented.
Journal ArticleDOI
Synthetic sites for transcription termination and a functional comparison with tryptophan operon termination sites in vitro
TL;DR: A model for termination involving separate but essential roles for the RNA hairpin and the stretch of uridines in the transcript is supported, supported bycription with the rpo203 polymerase and with ribonucleotide analogs.
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Tandem termination sites in the tryptophan operon of Escherichia coli
TL;DR: In vitro studies of DNA fragments carrying portions of the trp termination region demonstrate that RNA polymerase actually recognizes two different termination sites, contrasting with the observation that termination of transcription at the end of the tryptophan operon in vivo appears to require a rho-mediated interaction between trp t andtrp t'.
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Interactions between satellite bacteriophage P4 and its helpers
TL;DR: The helper dependence of satellite phage P4 superimposes an additional set of regulatory interactions on those required for the independent maintenance of P4 or its helpers, which promises to provide new insights into strategies for control of gene expression.
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The effects of hemodynamic shock and increased intra-abdominal pressure on bacterial translocation.
James M. Doty,Jun Oda,Rao R. Ivatury,Charles R. Blocher,Gail E. Christie,Jay A. Yelon,Harvey J. Sugerman +6 more
TL;DR: Hemorrhage followed by reperfusion and a subsequent insult of IAH caused significant GI mucosal acidosis, hypoperfusion, as well as systemic acidosis in Yorkshire swine, but changes did not appear to be associated with a significant bacterial translocation as judged by PCR measurements, tissue, or blood cultures.