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Galen Buckwalter

Researcher at University of Southern California

Publications -  6
Citations -  395

Galen Buckwalter is an academic researcher from University of Southern California. The author has contributed to research in topics: Epidemiology & Synaptic scaling. The author has an hindex of 6, co-authored 6 publications receiving 383 citations.

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Occupations with Exposure to Electromagnetic Fields: A Possible Risk Factor for Alzheimer's Disease

TL;DR: Analysis of data from three independent clinical series and controls indicates an association between working in occupations with probable medium to high exposure to extremely low frequency (< 300 Hz) electromagnetic fields and sporadic Alzheimer's disease.
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The frequently low cobalamin levels in dementia usually signify treatable metabolic, neurologic and electrophysiologic abnormalities

TL;DR: Cobalamin therapy improved 50–100% of the various types of abnormalities, although it did not improve cognitive function in the 13 demented patients, and frequent reversibility by cobalamin confirms that these defects indeed arise from cobalamine deficiency.
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Parental intrusiveness in adolescence recalled by women with a history of bulimia nervosa and comparison women.

TL;DR: These results support clinical observations of high levels of parental intrusiveness in the adolescent experiences of women who develop BN and provide further validation of the Parental Intrusiveness Rating Scale.
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Decreased duration of Ca(2+)-mediated plateau potentials in striatal neurons from aged rats.

TL;DR: The morphological findings suggest that dendritic regression in aged neurons may have reduced the number of Ca(2+) channels participating in plateau potential generation, but other mechanisms related to changes in the type of Ca2+ channel expressed and possible differences in their inactivation kinetics may also contribute to the age-related change in plateau possible duration.
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Age-related change in short-term synaptic plasticity intrinsic to excitatory striatal synapses.

TL;DR: Results imply an age‐dependent association between altered dendritic morphology and changes in synaptic plasticity, which may be due in part to a reduction in the readily releasable pool of synaptic vesicles.