G
Gary B. Quistad
Researcher at University of California, Berkeley
Publications - 103
Citations - 5050
Gary B. Quistad is an academic researcher from University of California, Berkeley. The author has contributed to research in topics: Methoprene & Metabolite. The author has an hindex of 35, co-authored 103 publications receiving 4809 citations.
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Journal ArticleDOI
Golden age of insecticide research: past, present, or future?
John E. Casida,Gary B. Quistad +1 more
TL;DR: Insecticide research, having passed through several Golden Ages, is now in a renaissance of integrating chemicals and biologicals for sustainable pest control with human safety.
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Organophosphate toxicology: safety aspects of nonacetylcholinesterase secondary targets.
John E. Casida,Gary B. Quistad +1 more
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Identification of an allatostatin from the tobacco hornworm Manduca sexta.
Steven J. Kramer,Anne Toschi,Christine Miller,Hiroshi Kataoka,Gary B. Quistad,Jorge P. Li,Robert L. Carney,David A. Schooley +7 more
TL;DR: A peptide that strongly inhibits juvenile hormone biosynthesis in vitro by the corpora allata from fifth-stadium larvae and adult females has been purified from extracts of heads of pharate adult M. sexta by a nine-step purification procedure and has no sequence similarity with any known neuropeptide from other organisms.
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Purification and characterization of two classes of neurotoxins from the funnel web spider, Agelenopsis aperta.
W S Skinner,Michael E. Adams,Gary B. Quistad,Hiroshi Kataoka,B J Cesarin,F. E. Enderlin,David A. Schooley +6 more
TL;DR: Two classes of paralytic toxins were isolated from the venom of Agelenopsis aperta and their chemical and larvicidal properties characterized and the secondary structure of one of these toxins (mu-Aga V) was investigated.
Journal ArticleDOI
Loss of neuropathy target esterase in mice links organophosphate exposure to hyperactivity.
Christopher J. Winrow,Christopher J. Winrow,Matthew L. Hemming,Duane M. Allen,Gary B. Quistad,John E. Casida,Carrolee Barlow,Carrolee Barlow +7 more
TL;DR: These studies show that genetic or chemical reduction of Nte activity results in a neurological phenotype of hyperactivity in mammals and indicate that EOPF toxicity occurs directly through inhibition of NTE without the requirement for Nte gain of function or aging.