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Gary Balian

Researcher at University of Virginia

Publications -  98
Citations -  5710

Gary Balian is an academic researcher from University of Virginia. The author has contributed to research in topics: Stromal cell & Mesenchymal stem cell. The author has an hindex of 43, co-authored 97 publications receiving 5390 citations. Previous affiliations of Gary Balian include University of Virginia Health System.

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Tendon: biology, biomechanics, repair, growth factors, and evolving treatment options.

TL;DR: Screening the genes expressed during tendon morphogenesis and determining the growth factors most crucial for tendon development will likely lead to treatment options that result in superior repair tissue and ultimately improved functional outcomes.
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Establishing human prostate cancer cell xenografts in bone: Induction of osteoblastic reaction by prostate‐specific antigen‐producing tumors in athymic and SCID/bg mice using LNCaP and lineage‐derived metastatic sublines

TL;DR: A PSA‐producing and osteoblastic human prostate cancer xenograft model in mice is established and whereas LNCaP cells injected intracardially failed to develop metastasis, C4‐2 cells injected similarly had the highest metastatic capability in SCID/bg mice.
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The Pathogenesis and Prevention of Steroid Induced Osteonecrosis

TL;DR: In this paper, the effects of a lipid lowering agent, lovastatin, on the prevention of steroid induced adipogenesis in vitro in cell culture, and on adipogenesis and osteonecrosis in vivo in chickens, were evaluated.
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Marrow stromal cells embedded in alginate for repair of osteochondral defects

TL;DR: Alginate induces the chondrogenic phenotype in marrow stromal cells in vitro, and possesses the necessary physical characteristics and handling properties to support cells and serve as a carrier to fill full-thickness osteochondral defects in vivo.
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Steroid-induced adipogenesis in a pluripotential cell line from bone marrow.

TL;DR: The data indicate that dexamethasone stimulates the differentiation of cells in bone-marrow stroma into adipocytes as well as the accumulation of fat in the marrow at the expense of expression of type-I collagen and osteocalcin mRNA, thereby suppressing differentiation into osteoblasts.