G
Gavin A. Scott
Researcher at University of Calgary
Publications - 19
Citations - 212
Gavin A. Scott is an academic researcher from University of Calgary. The author has contributed to research in topics: Working memory & Neurogenesis. The author has an hindex of 5, co-authored 18 publications receiving 127 citations. Previous affiliations of Gavin A. Scott include University of Saskatchewan & University of Ontario Institute of Technology.
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Journal ArticleDOI
Prospective Analysis of the Effects of Maternal Immune Activation on Rat Cytokines during Pregnancy and Behavior of the Male Offspring Relevant to Schizophrenia.
Brittney R. Lins,Jessica L. Hurtubise,Andrew J. Roebuck,Wendie N. Marks,Nadine K. Zabder,Gavin A. Scott,Quentin Greba,Wojciech Dawicki,Xiaobei Zhang,Christopher D. Rudulier,John R. Gordon,John G. Howland +11 more
TL;DR: Overall, while the offspring of polyI:C-treated rats displayed behavior abnormalities, maternal serum cytokines were not related to the long-term behavior changes in the offspring.
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Sex Difference in Cue Strategy in a Modified Version of the Morris Water Task: Correlations between Brain and Behaviour
TL;DR: The study revealed that males outperformed females overall during training trials, relied on the geometric cue when the platform was moved and showed significant correlations between entorhinal cortex thickness and spatial memory performance.
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Disrupted Neurogenesis in Germ-Free Mice: Effects of Age and Sex.
TL;DR: The results show that disruption of the gut microbiome affects hippocampal neurogenesis in an age- and sex-dependent manner and that these changes are also related to changes in the dentate gyrus functional network.
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Acute stress, but not corticosterone, facilitates acquisition of paired associates learning in rats using touchscreen-equipped operant conditioning chambers.
TL;DR: It is suggested that while acute stress enhances the acquisition of PAL, CORT alone does not, and this dissociation may be due to differences between these treatments and their ability to produce sufficient catecholamine release in the amygdala, a requirement for stress effects on memory.
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Performance of the trial-unique, delayed non-matching-to-location (TUNL) task depends on AMPA/Kainate, but not NMDA, ionotropic glutamate receptors in the rat posterior parietal cortex.
TL;DR: The results indicate that performance of the TUNL task depends on the PPC but that NMDA receptor signaling within this brain area is not necessary for intact performance.